Document Detail


Signaling meets chromatin during tissue regeneration in Drosophila.
MedLine Citation:
PMID:  16919444     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
As transcription programs become stabilized in fate-determined cells by progressive patterning of chromatin structures, cells lose their plasticity and the ability to freely modify their identity in response to changing developmental cues. By contrast, stem cells maintain this flexibility, enabling them to embark on different determination pathways. However, regeneration of tissue requires an exception because determined cells are forced to switch their transcription programs to reconstruct the missing tissue. In Drosophila, proliferating cells in the regenerating imaginal discs can even switch to a new disc identity. New studies show that the increased plasticity observed during regeneration results from the action of multiple signaling pathways on chromatin malleability, cell-cycle profiles, and expression of 'stemness' genes. Understanding how signaling pathways can integrate to switch determined cells into multipotent cells has a great medical potential, especially in the field of tissue engineering and remodeling.
Authors:
Cédric Maurange; Nara Lee; Renato Paro
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't; Review     Date:  2006-08-17
Journal Detail:
Title:  Current opinion in genetics & development     Volume:  16     ISSN:  0959-437X     ISO Abbreviation:  Curr. Opin. Genet. Dev.     Publication Date:  2006 Oct 
Date Detail:
Created Date:  2006-09-18     Completed Date:  2006-11-13     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  9111375     Medline TA:  Curr Opin Genet Dev     Country:  England    
Other Details:
Languages:  eng     Pagination:  485-9     Citation Subset:  IM    
Affiliation:
MRC National Institute for Medical Research, The Ridgeway, Mill Hill, London, NW7 1AA, UK.
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MeSH Terms
Descriptor/Qualifier:
Animals
Chromatin / genetics*
Drosophila / genetics*,  metabolism*
Gene Expression Regulation
Regeneration / genetics*,  physiology*
Signal Transduction*
Stem Cells / metabolism
Chemical
Reg. No./Substance:
0/Chromatin

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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