Document Detail


Sickle cell biomechanics.
MedLine Citation:
PMID:  20455701     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
As the predominant cell type in blood, red blood cells (RBCs) and their biomechanical properties largely determine the rheological and hemodynamic behavior of blood in normal and disease states. In sickle cell disease (SCD), mechanically fragile, poorly deformable RBCs contribute to impaired blood flow and other pathophysiological aspects of the disease. The major underlying cause of this altered blood rheology and hemodynamics is hemoglobin S (HbS) polymerization and RBC sickling under deoxygenated conditions. This review discusses the characterization of the biomechanical properties of sickle RBCs and sickle blood as well as their implications toward a better understanding of the pathophysiology of the disease.
Authors:
Gilda A Barabino; Manu O Platt; Dhananjay K Kaul
Publication Detail:
Type:  Journal Article; Review    
Journal Detail:
Title:  Annual review of biomedical engineering     Volume:  12     ISSN:  1545-4274     ISO Abbreviation:  Annu Rev Biomed Eng     Publication Date:  2010 Aug 
Date Detail:
Created Date:  2010-07-12     Completed Date:  2010-10-21     Revised Date:  2011-09-14    
Medline Journal Info:
Nlm Unique ID:  100883581     Medline TA:  Annu Rev Biomed Eng     Country:  United States    
Other Details:
Languages:  eng     Pagination:  345-67     Citation Subset:  IM    
Affiliation:
The Wallace H. Coulter Department of Biomedical Engineering, Georgia Institute of Technology and Emory University, Atlanta, Georgia 30332, USA. barabino@gatech.edu
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Anemia, Sickle Cell / blood*,  physiopathology*
Animals
Biomechanics
Cats
Constriction, Pathologic
Disease Models, Animal
Erythrocyte Deformability*
Erythrocyte Membrane / physiology
Erythrocytes, Abnormal / pathology,  physiology*
Hemodynamics / physiology
Hemoglobin, Sickle / metabolism
Humans
Rabbits
Rats
Grant Support
ID/Acronym/Agency:
R01 HL070047-07/HL/NHLBI NIH HHS; R01 HL070047-08/HL/NHLBI NIH HHS
Chemical
Reg. No./Substance:
0/Hemoglobin, Sickle

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Translational research on Trichoderma: from 'omics to the field.
Next Document:  Gene expression profiling of breast cancer cell lines in response to soy isoflavones using a pangeno...