| Short-term inhibition of cardiac cellular autophagy by isoproterenol. | |
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MedLine Citation:
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PMID: 3443985 Owner: NLM Status: MEDLINE |
Abstract/OtherAbstract:
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It is shown in the present study by means of quantitative electron microscopy that the volume fraction of autophagic vacuoles is significantly reduced to about half the control value within a short time interval (about 10 min) after low dose administration of isoproterenol. The data suggest that stimulation of cardiac work is accompanied by an immediate anticatabolic reaction which can be assumed to represent an early contribution to the adaptative increase in myocardial mass known to result from long-term stimulation of cardiac work. |
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Authors:
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U Pfeifer; J Föhr; W Wilhelm; J Dämmrich |
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Publication Detail:
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Type: Journal Article; Research Support, Non-U.S. Gov't |
Journal Detail:
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Title: Journal of molecular and cellular cardiology Volume: 19 ISSN: 0022-2828 ISO Abbreviation: J. Mol. Cell. Cardiol. Publication Date: 1987 Dec |
Date Detail:
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Created Date: 1988-05-04 Completed Date: 1988-05-04 Revised Date: 2006-11-15 |
Medline Journal Info:
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Nlm Unique ID: 0262322 Medline TA: J Mol Cell Cardiol Country: ENGLAND |
Other Details:
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Languages: eng Pagination: 1179-84 Citation Subset: IM |
Affiliation:
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Department of Pathology, University of Würzburg, Germany. |
Export Citation:
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APA/MLA Format Download EndNote Download BibTex |
| MeSH Terms | |
Descriptor/Qualifier:
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Animals Autophagy / drug effects* Heart / drug effects* Heart Ventricles / ultrastructure Isoproterenol / pharmacology* Male Phagocytosis / drug effects* Rats Time Factors |
| Chemical | |
Reg. No./Substance:
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7683-59-2/Isoproterenol |
From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine
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