Document Detail


Sex-related splenocyte function in a murine model of allergic asthma.
MedLine Citation:
PMID:  18498415     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
BACKGROUND: The prevalence and severity of asthma are higher among boys than girls, but the ratios are reversed after puberty. These observations strongly suggest that sex hormones have a role in the pathogenesis of the disease. However, the mechanisms underlying the gender differences in asthma are not fully understood. OBJECTIVE: The aim of this study was to investigate sex differences in allergic inflammation in terms of immune function. METHODS: Male and female C57BL/6 mice were sensitized and challenged with ovalbumin (OVA). OVA-specific IgE in serum and airway inflammation were compared between sexes. Splenocytes from OVA-sensitized male or female donor mice were transferred to male or female naïve recipient mice. Subsequently, the recipient mice were challenged, followed by the evaluation of OVA-specific IgE and airway inflammation. Cytokines secreted from splenocytes of the sensitized mice were measured. RESULTS: The levels of OVA-specific IgE and the allergen-induced airway inflammation were higher in female than in the male mice. The contents of T-helper type 2 (Th2) cytokines, IL-4, IL-5 and IL-13, in the bronchoalveolar lavage fluid from female mice were higher than those from male mice. The airway inflammation in female recipients transferred with splenocytes from female donors was more severe than that in any other combination of recipients and donors. Splenocytes from the sensitized female mice produced more of the Th2 cytokine, IL-5, than those from the sensitized male mice upon stimulation with OVA. CONCLUSION: Our findings suggest that the sex difference in allergic airway inflammation may be attributable to the sex difference in not only the hormonal environment but also in the immune cells themselves.
Authors:
K Okuyama; K Wada; J Chihara; M Takayanagi; I Ohno
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2008-05-21
Journal Detail:
Title:  Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology     Volume:  38     ISSN:  1365-2222     ISO Abbreviation:  Clin. Exp. Allergy     Publication Date:  2008 Jul 
Date Detail:
Created Date:  2008-08-11     Completed Date:  2008-10-27     Revised Date:  2008-11-21    
Medline Journal Info:
Nlm Unique ID:  8906443     Medline TA:  Clin Exp Allergy     Country:  England    
Other Details:
Languages:  eng     Pagination:  1212-9     Citation Subset:  IM    
Affiliation:
Department of Pathophysiology, Tohoku Pharmaceutical University, Sendai, Japan.
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MeSH Terms
Descriptor/Qualifier:
Animals
Asthma / immunology*,  metabolism
Bronchoalveolar Lavage Fluid / cytology,  immunology*
Eosinophils / immunology,  metabolism
Female
Immunoglobulin E / blood*
Inflammation / immunology,  metabolism
Interferon-gamma / immunology,  metabolism
Interleukin-13 / biosynthesis,  immunology
Interleukin-4 / biosynthesis,  immunology
Interleukin-5 / biosynthesis,  immunology
Male
Mice
Mice, Inbred C57BL
Ovalbumin / immunology*
Sex Characteristics*
Chemical
Reg. No./Substance:
0/Interleukin-13; 0/Interleukin-5; 207137-56-2/Interleukin-4; 37341-29-0/Immunoglobulin E; 82115-62-6/Interferon-gamma; 9006-59-1/Ovalbumin

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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