Document Detail


Senescent phenotype can be reversed by reduction of caveolin status.
MedLine Citation:
PMID:  12730243     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Hyporesponsiveness to growth factors is one of the fundamental characteristics of senescent cells. We previously reported that the up-regulation of caveolin attenuates the growth factor response and the subsequent downstream signal cascades in senescent human diploid fibroblasts. Therefore, in the present experiment, we investigated the modulation of caveolin status in senescent cells to determine the effect of caveolin on mitogenic signaling efficiency and cell cycling. We reduced the level of caveolin-1 in senescent human diploid fibroblasts using its antisense oligonucleotides and small interfering RNA, and this resulted in the restoration of normal growth factor responses such as the increased phosphorylation of Erk, the nuclear translocation of p-Erk, and the subsequent activation of p-Elk upon epidermal growth factor stimulation. Moreover, DNA synthesis and the re-entry of senescent cells into cell cycle were resumed upon epidermal growth factor stimulation concomitantly with decreases in p53 and p21. Taken together, we conclude that the loss of mitogenic signaling in senescent cells is strongly related to their elevated levels of caveolin-1 and that the functional recovery of senescent cells at least in the terms of growth factor responsiveness and cell cycle entry might be achieved simply by lowering the caveolin level.
Authors:
Kyung A Cho; Sung Jin Ryu; Jeong Soo Park; Ik Soon Jang; Jeong Soo Ahn; Kyung Tae Kim; Sang Chul Park
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2003-05-01
Journal Detail:
Title:  The Journal of biological chemistry     Volume:  278     ISSN:  0021-9258     ISO Abbreviation:  J. Biol. Chem.     Publication Date:  2003 Jul 
Date Detail:
Created Date:  2003-07-21     Completed Date:  2003-08-26     Revised Date:  2009-11-19    
Medline Journal Info:
Nlm Unique ID:  2985121R     Medline TA:  J Biol Chem     Country:  United States    
Other Details:
Languages:  eng     Pagination:  27789-95     Citation Subset:  IM    
Affiliation:
The Aging and Apoptosis Research Center, Department of Biochemistry and Molecular Biology, Seoul National University College of Medicine, Seoul 110-799, Korea.
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MeSH Terms
Descriptor/Qualifier:
Blotting, Western
Caveolin 1
Caveolins / metabolism*
Cell Aging*
Cell Cycle
Cell Division
Cells, Cultured
DNA / biosynthesis,  metabolism
Down-Regulation
Epidermal Growth Factor / metabolism
Fibroblasts / metabolism
Humans
Immunohistochemistry
Microscopy, Electron
Microscopy, Fluorescence
Mitogen-Activated Protein Kinases / metabolism
Oligonucleotides, Antisense / metabolism
Phenotype
Proto-Oncogene Proteins p21(ras) / metabolism
RNA, Small Interfering / metabolism
Signal Transduction
Subcellular Fractions / metabolism
Time Factors
Transfection
Tumor Suppressor Protein p53 / metabolism
Up-Regulation
Chemical
Reg. No./Substance:
0/CAV1 protein, human; 0/Caveolin 1; 0/Caveolins; 0/Oligonucleotides, Antisense; 0/RNA, Small Interfering; 0/Tumor Suppressor Protein p53; 62229-50-9/Epidermal Growth Factor; 9007-49-2/DNA; EC 2.7.11.24/Mitogen-Activated Protein Kinases; EC 3.6.5.2/HRAS protein, human; EC 3.6.5.2/Proto-Oncogene Proteins p21(ras)

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