Document Detail


Scavenging free radicals improves endothelial dysfunction in human coronary arteries in vivo.
MedLine Citation:
PMID:  20676959     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The effect of a novel free radical scavenger, edaravone, on coronary endothelial dysfunction was examined in patients who had no significant stenosis of coronary arteries, to elucidate the role of free radicals on coronary endothelial dysfunction. The coronary blood flow (CBF) responses to acetylcholine (ACh) were measured by quantitative coronary arteriography and the intracoronary Doppler technique before and after the administration of edaravone. Twenty-four patients were divided into two groups on the basis of CBF responses to ACh; those with "attenuated" (%Delta CBF < 300%, n = 12) and "normal" (%Delta CBF > 300%, n = 12) flow responses. An intracoronary infusion of edaravone significantly improved ACh-induced increases in CBF in patients with attenuated flow responses; however, edaravone had no effect in those with normal flow responses (36.8% +/- 7.3% vs 0.0% +/- 5.1%, P < 0.01). The plasma levels of nitric oxide compounds (NOx) in the attenuated response group were lower than those in the normal group (35.7 +/- 2.3 vs 49.4 +/- 6.2 muM, P < 0.01) and correlated with the magnitude of CBF improvement by edaravone (r = 0.566, P < 0.01). The plasma level of malondialdehyde and 4-hydroxynonenal, which indicates the level of oxidative stress, in the attenuated group was higher than that in the normal group (6.9 +/- 0.9 vs 3.3 +/- 0.5 muM, P < 0.01) and correlated with the magnitude of CBF improvement by edaravone (r = 0.854, P < 0.01). A free radical scavenger improved the ACh-induced CBF response in patients with coronary atherosclerosis in vivo. Therefore, the current results suggest that scavenging free radicals has a beneficial effect for patients with coronary endothelial dysfunction.
Authors:
Jun-ichi Oyama; Shinji Satoh; Nobuhiro Suematsu; Toshiaki Kadokami; Toyoki Maeda; Masahiro Sugano; Naoki Makino
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Publication Detail:
Type:  Journal Article     Date:  2010-07-31
Journal Detail:
Title:  Heart and vessels     Volume:  25     ISSN:  1615-2573     ISO Abbreviation:  Heart Vessels     Publication Date:  2010 Sep 
Date Detail:
Created Date:  2010-08-02     Completed Date:  2010-11-22     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  8511258     Medline TA:  Heart Vessels     Country:  Japan    
Other Details:
Languages:  eng     Pagination:  379-85     Citation Subset:  IM    
Affiliation:
Department of Cardiovascular, Respiratory, and Geriatric Medicine, Kyushu University Hospital at Beppu and Medical Institute of Bioregulation, Kyushu University, Oita, Japan. joyama@tsurumi.beppu.kyushu-u.ac.jp
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MeSH Terms
Descriptor/Qualifier:
Acetylcholine / administration & dosage
Aged
Aldehydes / blood
Antipyrine / administration & dosage,  analogs & derivatives*
Blood Flow Velocity / drug effects
Coronary Angiography
Coronary Circulation / drug effects*
Coronary Vessels / drug effects*,  metabolism,  physiopathology,  ultrasonography
Dose-Response Relationship, Drug
Echocardiography, Doppler
Endothelium, Vascular / drug effects*,  metabolism,  physiopathology
Female
Free Radical Scavengers / administration & dosage*
Free Radicals / metabolism*
Humans
Infusions, Intra-Arterial
Isosorbide Dinitrate / administration & dosage
Japan
Male
Malondialdehyde / blood
Nitric Oxide / blood
Oxidative Stress / drug effects*
Vasodilation / drug effects*
Vasodilator Agents / administration & dosage
Chemical
Reg. No./Substance:
0/Aldehydes; 0/Free Radical Scavengers; 0/Free Radicals; 0/Vasodilator Agents; 10102-43-9/Nitric Oxide; 29343-52-0/4-hydroxy-2-nonenal; 51-84-3/Acetylcholine; 542-78-9/Malondialdehyde; 60-80-0/Antipyrine; 87-33-2/Isosorbide Dinitrate; 89-25-8/phenylmethylpyrazolone

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