| SOS - too many signals for systemic acquired resistance? | |
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MedLine Citation:
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PMID: 22749315 Owner: NLM Status: Publisher |
Abstract/OtherAbstract:
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Following pathogen infection, activation of systemic acquired resistance (SAR) in uninfected tissues requires transmission of a signal(s) from the infected tissue via the vasculature. Several candidates for this long-distance signal have been identified, including methyl salicylate (MeSA), an SFD1/GLY1-derived glycerol-3-phosphate (G3P)-dependent signal, the lipid-transfer protein DIR1, the dicarboxylic acid azelaic acid (AzA), the abietane diterpenoid dehydroabietinal (DA), jasmonic acid (JA), and the amino acid-derivative pipecolic acid (Pip). Some of these signals work cooperatively to activate SAR and/or regulate MeSA metabolism. However, Pip appears to activate SAR via an independent pathway that may impinge on these other signaling pathway(s) during de novo salicylic acid (SA) biosynthesis in the systemic tissue. Thus, a complex web of cross-interacting signals appears to activate SAR. |
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Authors:
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D'Maris Amick Dempsey; Daniel F Klessig |
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Publication Detail:
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Type: JOURNAL ARTICLE Date: 2012-6-28 |
Journal Detail:
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Title: Trends in plant science Volume: - ISSN: 1878-4372 ISO Abbreviation: - Publication Date: 2012 Jun |
Date Detail:
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Created Date: 2012-7-3 Completed Date: - Revised Date: - |
Medline Journal Info:
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Nlm Unique ID: 9890299 Medline TA: Trends Plant Sci Country: - |
Other Details:
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Languages: ENG Pagination: - Citation Subset: - |
Copyright Information:
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Copyright © 2012 Elsevier Ltd. All rights reserved. |
Affiliation:
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Boyce Thompson Institute for Plant Research, Ithaca, NY 14853, USA. |
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From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine
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