Document Detail


S-phase reduction in T47D human breast cancer epithelial cells induced by an S100P antisense-retroviral construct.
MedLine Citation:
PMID:  17273741     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
S100P is expressed in several malignant neoplasms. It was previously demonstrated that S100P is involved in the very early stages of breast carcinogenesis. In the present study we used a retrovirus-mediated transfer of antisense-S100P in order to check whether the decrease in expression of this protein could lead to alterations in the cell cycle of epithelial cells of human breast cancer. The T47D breast carcinoma cell line, a human breast epithelial cell that expresses high levels of S100P, was a tool used in this study to investigate the alteration in cell cycle induced by a retrovirus-mediated transfer of antisense-S100P. First we used the real-time PCR technique to quantify the gene expression. The results showed a reduction of 63% of expression within the T47D-S100P-A/S infected population compared with control T47D-LXSN clones. To determine the impact of the S100P antisense technique on protein expression in T47D cells, we performed immunofluorescence staining and analyzed the resulting images using a confocal microscope. The images showed much less pronounced antibody marking of the S100P protein in the T47D-S100P-A/S compared with control cells. To evaluate whether the antisense approach caused any alteration in the cell cycle, we concluded the study with flow cytometric analysis of the cell distribution. Our findings indicated that, in our model, S100P-antisense cells showed a 23% reduction of cells at the S-phase. Using transduction techniques with an S100P antisense-retroviral construct we were able to demonstrate a significant reduction in S-phase of the T47D cell cycle. To the best of our knowledge, this is the first time that an antisense approach has been used against S100P mRNA in breast cancer epithelial cells. The results showed here seem to further classify S100P as a protein that might be involved in the cell cycle imbalance observed during breast carcinogenesis.
Authors:
Bettina Beissel; Ismael D C G Silva; João B Pesquero; Jose Russo; Nestor Schor; Maria Helena Bellini
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Oncology reports     Volume:  17     ISSN:  1021-335X     ISO Abbreviation:  Oncol. Rep.     Publication Date:  2007 Mar 
Date Detail:
Created Date:  2007-02-02     Completed Date:  2007-05-02     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  9422756     Medline TA:  Oncol Rep     Country:  Greece    
Other Details:
Languages:  eng     Pagination:  611-5     Citation Subset:  IM    
Affiliation:
Centro de Química e Meio Ambiente, IPEN/CNEN-SP, 04039-032 São Paulo, SP, Brazil.
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MeSH Terms
Descriptor/Qualifier:
Breast Neoplasms / metabolism*
Calcium-Binding Proteins / antagonists & inhibitors*,  genetics,  metabolism
Cell Cycle / physiology*
Cell Line, Tumor
Epithelial Cells / metabolism*
Female
Flow Cytometry
Fluorescent Antibody Technique
Gene Expression
Humans
Microscopy, Confocal
Neoplasm Proteins / antagonists & inhibitors*,  genetics,  metabolism
RNA, Antisense*
Retroviridae / genetics
Reverse Transcriptase Polymerase Chain Reaction
Chemical
Reg. No./Substance:
0/Calcium-Binding Proteins; 0/Neoplasm Proteins; 0/RNA, Antisense; 0/S100P protein, human

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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