Document Detail


Roles of gene transcription and PKA subtype activation in maturation of murine oocytes.
MedLine Citation:
PMID:  12052234     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The aims of this study were to examine the role of transcription and the coincident involvement of type I and type II protein kinase A (PKA) in the resumption of meiosis in murine cumulus-oocyte complexes (COCs) using the transcriptional inhibitors 5,6-dichloro-1-beta-D-ribofuranosylbenzimidazole (DRB) and alpha-amanitin. The first series of experiments was designed to: (i) characterize the role of transcription in gonadotrophin-mediated and spontaneous maturation of murine oocytes; (ii) examine the roles of specific gonadotrophins (FSH versus hCG) and cumulus cells in transcriptionally mediated oocyte maturation; and (iii) determine the reversibility of the transcriptional arrest of meiosis. In the presence of FSH, transcriptional inhibitors arrested germinal vesicle breakdown (GVBD) (DRB: 2 +/- 2% and control: 76 +/- 2%; alpha-amanitin: 4 +/- 4% and control: 70 +/- 4%). Furthermore, cumulus cells were required for transcriptional inhibitors to arrest GVBD (DRB with cumulus cells: 0 +/- 15%; DRB without cumulus cells: 94 +/- 13%; alpha-amanitin with cumulus cells: 15 +/- 2%; alpha-amanitin without cumulus cells: 99 +/- 2%). Thus, in mice, FSH-mediated GVBD uses a transcriptional mechanism, which probably occurs within the cumulus cell compartment. In a second series of experiments, the role of transcription in mediating the resumption of meiosis after activation of either type I or type II PKA was examined. Activation of type I PKA in murine COCs resulted in an arrest of GVBD that was independent of a transcriptional event (with DRB: 7 +/- 9% GVBD; without DRB: 11 +/- 9% GVBD). In contrast, activation of type II PKA resulted in a resumption of meiosis, which required the occurrence of gene transcription (with DRB: 12 +/- 9% GVBD; without DRB: 80 +/- 9% GVBD). As FSH binding to cumulus cells activates the PKA second messenger system, our results indicate that, in cultured murine COCs, FSH binding to cumulus cells results in the activation of type II PKA, which, in turn, mediates a downstream transcriptional event required for the initiation of GVBD.
Authors:
K F Rodriguez; R M Petters; A E Crosier; C E Farin
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Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  Reproduction (Cambridge, England)     Volume:  123     ISSN:  1470-1626     ISO Abbreviation:  Reproduction     Publication Date:  2002 Jun 
Date Detail:
Created Date:  2002-06-07     Completed Date:  2002-07-19     Revised Date:  2007-11-15    
Medline Journal Info:
Nlm Unique ID:  100966036     Medline TA:  Reproduction     Country:  England    
Other Details:
Languages:  eng     Pagination:  799-806     Citation Subset:  IM    
Affiliation:
Department of Animal Science, North Carolina State University, 231B Polk Hall, Raleigh, NC 27695-7621, USA.
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MeSH Terms
Descriptor/Qualifier:
Amanitins / pharmacology
Animals
Cells, Cultured
Chorionic Gonadotropin / pharmacology
Cyclic AMP-Dependent Protein Kinase Type II
Cyclic AMP-Dependent Protein Kinases / antagonists & inhibitors,  metabolism*
Dichlororibofuranosylbenzimidazole / pharmacology
Enzyme Activation / physiology*
Enzyme Inhibitors / pharmacology
Female
Follicle Stimulating Hormone / pharmacology
Isoenzymes / metabolism
Mice
Mice, Inbred Strains
Oogenesis / physiology*
Transcription, Genetic / drug effects,  physiology*
Chemical
Reg. No./Substance:
0/Amanitins; 0/Chorionic Gonadotropin; 0/Enzyme Inhibitors; 0/Isoenzymes; 53-85-0/Dichlororibofuranosylbenzimidazole; 9002-68-0/Follicle Stimulating Hormone; EC 2.7.11.11/Cyclic AMP-Dependent Protein Kinase Type II; EC 2.7.11.11/Cyclic AMP-Dependent Protein Kinases

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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