Document Detail


Role of toxic oxygen products from phagocytic cells in tissue injury.
MedLine Citation:
PMID:  6308973     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The generation of toxic oxygen products from activated phagocytic cells represents an important pathogenic mechanism in tissue damage associated with inflammatory reactions that are characterized by the involvement of neutrophils or other phagocytic cells. The production of these toxic products from activated phagocytic cells is related to receptor activation on the surface of the cells. Most emphasis has been placed on studies in the lung where the production of these toxic metabolites by phagocytic cells, either in the vasculature or in the airway compartment of the lungs, has been demonstrated to result in endothelial cell injury or damage and destruction of alveolar lining cells. In some cases these reactions are progressive, resulting in parenchymal collapse and fibrosis. Naturally occurring protective factors have been demonstrated both within cells and in the plasma and interstitial fluid compartments. Although there is increasing evidence that in the shock syndrome complement activation products and toxic oxygen metabolites from activated leukocytes play important roles, the most direct evidence for related mechanisms comes in detailed studies of immune complex induced injury where a role for both O-2 and H2O2 has been clearly demonstrated. These facts indicate that toxic oxygen metabolites from activated phagocytic cells play important roles in a variety of pathological situations, and have provided new insights into intervention therapy.
Authors:
P A Ward
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Publication Detail:
Type:  Journal Article; Review    
Journal Detail:
Title:  Advances in shock research     Volume:  10     ISSN:  0195-878X     ISO Abbreviation:  Adv Shock Res     Publication Date:  1983  
Date Detail:
Created Date:  1983-09-09     Completed Date:  1983-09-09     Revised Date:  2005-11-17    
Medline Journal Info:
Nlm Unique ID:  7908298     Medline TA:  Adv Shock Res     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  27-34     Citation Subset:  IM    
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MeSH Terms
Descriptor/Qualifier:
Animals
Antigen-Antibody Complex / physiology
Arthus Reaction / immunology,  pathology
Complement C5 / physiology
Complement C5a
Free Radicals
Hydrogen Peroxide / metabolism
Inflammation / immunology*,  pathology
Neutrophils / immunology*
Oxygen / metabolism*
Phagocytosis*
Rats
Superoxide Dismutase / physiology
Superoxides / biosynthesis
Tetradecanoylphorbol Acetate / pharmacology
Chemical
Reg. No./Substance:
0/Antigen-Antibody Complex; 0/Complement C5; 0/Free Radicals; 11062-77-4/Superoxides; 16561-29-8/Tetradecanoylphorbol Acetate; 7722-84-1/Hydrogen Peroxide; 7782-44-7/Oxygen; 80295-54-1/Complement C5a; EC 1.15.1.1/Superoxide Dismutase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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