Document Detail


Role of pancreatic-derived factor in type 2 diabetes: evidence from pancreatic β cells and liver.
MedLine Citation:
PMID:  22300596     Owner:  NLM     Status:  In-Data-Review    
Abstract/OtherAbstract:
Pancreatic-derived factor (PANDER) is a cytokine-like protein that is highly expressed in pancreatic islets. In vitro, PANDER pretreatment or viral-mediated overexpression promotes apoptosis of islet β cells. Under conditions of insulin resistance, chronic hyperglycemia potently activates PANDER expression and stimulates the cosecretion of insulin and PANDER in β cells. PANDER binds to the liver cell membrane and induces insulin resistance, resulting in increased gluconeogenesis. Recently, PANDER was found to be expressed in rodent and human liver, and its expression is increased in the liver of diabetic mice and rats. Hepatic overexpression of PANDER promotes lipogenesis in the liver and induces insulin resistance in C57BL/6 mice, whereas the inactivation of hepatic PANDER markedly reduces steatosis, insulin resistance, and hyperglycemia in db/db mice. PANDER deficiency protects mice from high-fat-diet-induced hyperglycemia by decreasing gluconeogenesis in the liver. In summary, PANDER plays an important role in the progression of type 2 diabetes by negatively regulating islet β-cell function and insulin sensitivity in the liver.
Authors:
Chunjiong Wang; Brant R Burkhardt; Youfei Guan; Jichun Yang
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Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  Nutrition reviews     Volume:  70     ISSN:  1753-4887     ISO Abbreviation:  Nutr. Rev.     Publication Date:  2012 Feb 
Date Detail:
Created Date:  2012-02-03     Completed Date:  -     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  0376405     Medline TA:  Nutr Rev     Country:  United States    
Other Details:
Languages:  eng     Pagination:  100-6     Citation Subset:  IM    
Copyright Information:
© 2012 International Life Sciences Institute.
Affiliation:
Department of Physiology and Pathophysiology, Key Laboratory of Molecular Cardiovascular Science, Peking University Health Science Center, Beijing 100191, China Department of Cell Biology, Microbiology and Molecular Biology, University of South Florida, Tampa, Florida, USA.
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