Document Detail


Role of microRNA pathway in mental retardation.
MedLine Citation:
PMID:  17982588     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Deficits in cognitive functions lead to mental retardation (MR). Understanding the genetic basis of inherited MR has provided insights into the pathogenesis of MR. Fragile X syndrome is one of the most common forms of inherited MR, caused by the loss of functional Fragile X Mental Retardation Protein (FMRP). MicroRNAs (miRNAs) are endogenous, single-stranded RNAs between 18 and 25 nucleotides in length, which have been implicated in diversified biological pathways. Recent studies have linked the miRNA pathway to fragile X syndrome. Here we review the role of the miRNA pathway in fragile X syndrome and discuss its implication in MR in general.
Authors:
Abrar Qurashi; Shuang Chang; Jin Peng
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Publication Detail:
Type:  Journal Article; Review     Date:  2007-11-02
Journal Detail:
Title:  TheScientificWorldJournal     Volume:  7     ISSN:  1537-744X     ISO Abbreviation:  ScientificWorldJournal     Publication Date:  2007  
Date Detail:
Created Date:  2007-11-05     Completed Date:  2007-12-06     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  101131163     Medline TA:  ScientificWorldJournal     Country:  England    
Other Details:
Languages:  eng     Pagination:  146-54     Citation Subset:  IM    
Affiliation:
Department of Human Genetics, Emory University School of Medicine, Atlanta, GA, USA.
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MeSH Terms
Descriptor/Qualifier:
Animals
Biogenesis
Fragile X Mental Retardation Protein / genetics
Humans
Mental Retardation / genetics*
MicroRNAs / genetics*
Neurons / metabolism
Chemical
Reg. No./Substance:
0/MicroRNAs; 139135-51-6/Fragile X Mental Retardation Protein

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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