Document Detail


Role of hypoxic pulmonary vasoconstriction in pulmonary gas exchange and blood flow distribution. 2. Pathophysiology.
MedLine Citation:
PMID:  7930036     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
In this review, the second of a two part series, the analytic techniques introduced in the first part are applied to a broad range of pulmonary pathophysiologic conditions. The contributions of hypoxic pulmonary vasoconstriction to both homeostasis and pathophysiology are quantitated for atelectasis, pneumonia, sepsis, pulmonary embolism, chronic obstructive pulmonary disease and adult respiratory distress syndrome. For each disease state the influence of principle variables, including inspired oxygen concentration, cardiac output and severity of pathology are explored and the actions of selected drugs including inhaled nitric oxide and infused vasodilators are illustrated. It is concluded that hypoxic pulmonary vasoconstriction is often a critical determinant of hypoxemia and/or pulmonary hypertension. Furthermore this analysis demonstrates the value of computer simulation to reveal which of the many variables are most responsible for pathophysiologic results.
Authors:
B E Marshall; C W Hanson; F Frasch; C Marshall
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Publication Detail:
Type:  Journal Article; Research Support, U.S. Gov't, P.H.S.; Review    
Journal Detail:
Title:  Intensive care medicine     Volume:  20     ISSN:  0342-4642     ISO Abbreviation:  Intensive Care Med     Publication Date:  1994 May 
Date Detail:
Created Date:  1994-11-02     Completed Date:  1994-11-02     Revised Date:  2008-11-21    
Medline Journal Info:
Nlm Unique ID:  7704851     Medline TA:  Intensive Care Med     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  379-89     Citation Subset:  IM    
Affiliation:
Center for Anesthesia Research, University of Pennsylvania School of Medicine, Philadelphia 19104.
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MeSH Terms
Descriptor/Qualifier:
Anoxia / physiopathology*
Humans
Lung Diseases, Obstructive / physiopathology
Pneumonia / physiopathology
Pulmonary Artery / physiology*
Pulmonary Atelectasis / physiopathology
Pulmonary Circulation / physiology*
Pulmonary Gas Exchange / physiology*
Pulmonary Veins / physiology*
Respiratory Distress Syndrome, Adult / physiopathology
Vasoconstriction / physiology*
Grant Support
ID/Acronym/Agency:
GM29628/GM/NIGMS NIH HHS

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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