Document Detail


Role of endoplasmic reticulum stress in cystic fibrosis-related airway inflammatory responses.
MedLine Citation:
PMID:  21030518     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Chronic airway infection and inflammation are hallmarks of cystic fibrosis (CF) pulmonary disease. The altered airway environment resulting from infection and inflammation can affect the innate defense of the airway epithelia. Luminal bacterial and inflammatory stimuli trigger an adaptation in human airway epithelia, characterized by a hyperinflammatory response to inflammatory mediators, which is mediated by an expansion of the endoplasmic reticulum (ER) and its Ca(2+) stores. Recent studies demonstrated that a form of ER stress, the unfolded protein response (UPR), is activated in airway epithelia by bacterial infection-induced airway inflammation. UPR-dependent signaling is responsible for the ER Ca(2+) store expansion-mediated amplification of airway inflammatory responses. These studies highlight the functional importance of the UPR in airway inflammation and suggest that targeting the UPR may be a therapeutic strategy for airway diseases typified by chronic inflammation. This article reviews the contribution of airway epithelia to airway inflammatory responses, discusses how expansion of the ER Ca(2+) stores in inflamed airway epithelia contributes to airway inflammation, describes the functional role of the UPR in these processes, and discusses how UPR activation might be relevant for CF airways inflammatory disease.
Authors:
Carla M P Ribeiro; Richard C Boucher
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Review    
Journal Detail:
Title:  Proceedings of the American Thoracic Society     Volume:  7     ISSN:  1943-5665     ISO Abbreviation:  Proc Am Thorac Soc     Publication Date:  2010 Nov 
Date Detail:
Created Date:  2010-10-29     Completed Date:  2011-03-17     Revised Date:  2011-11-01    
Medline Journal Info:
Nlm Unique ID:  101203596     Medline TA:  Proc Am Thorac Soc     Country:  United States    
Other Details:
Languages:  eng     Pagination:  387-94     Citation Subset:  IM    
Affiliation:
Cystic Fibrosis/Pulmonary Research and Treatment Center and the Department of Medicine, University of North Carolina at Chapel Hill, Chapel Hill, North Carolina, USA. carla_ribeiro@med.unc.edu
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MeSH Terms
Descriptor/Qualifier:
Calcium / metabolism*
Cystic Fibrosis / metabolism,  physiopathology*
Endoplasmic Reticulum / metabolism*
Humans
Inflammation / metabolism*
Signal Transduction
Unfolded Protein Response
Grant Support
ID/Acronym/Agency:
HL34322/HL/NHLBI NIH HHS
Chemical
Reg. No./Substance:
7440-70-2/Calcium

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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