Document Detail


Role of constitutive nitric oxide synthase S-nitrosylation in Helicobacter pylori-induced gastric mucosal cell apoptosis: effect of ghrelin.
MedLine Citation:
PMID:  20596895     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Infection with H. pylori is a primary factor in the etiology of gastric disease, and the excessive NO generation and a massive rise in apoptosis are well recognized features that characterize the mucosal inflammatory responses to the bacterium and its lipopolysaccharide (LPS). Here, we report that H. pylori LPS-induced enhancement in gastric mucosal cell apoptosis and NO generation was associated with the suppression in constitutive nitric oxide synthase (cNOS) activity and a marked up-regulation in the activity of inducible nitric oxide synthase (iNOS). Further, we demonstrate that the detrimental effect of the LPS on cNOS was manifested in the enzyme protein S-nitrosylation, that was susceptible to suppression by iNOS inhibitor, 1400W. Moreover, we show that the countering effect of peptide hormone, ghrelin, on the LPS-induced changes in apoptosis and cNOS activity was reflected in the loss in cNOS S-nitrosylation and the increase in the enzyme phosphorylation. These findings demonstrate that the disturbances in gastric mucosal NO generation system caused by H. pylori result from the iNOS-derived NO suppression of cNOS activation through S-nitrosylation. We also report that ghrelin protection against H. pylori-induced gastric mucosal proapoptotic events involves cNOS activation manifested by the increase in enzyme protein phosphorylation and a decrease in its S-nitrosylation.
Authors:
B L Slomiany; A Slomiany
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Publication Detail:
Type:  Journal Article     Date:  2010-07-02
Journal Detail:
Title:  Inflammopharmacology     Volume:  18     ISSN:  1568-5608     ISO Abbreviation:  Inflammopharmacology     Publication Date:  2010 Oct 
Date Detail:
Created Date:  2010-09-20     Completed Date:  2010-12-28     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  9112626     Medline TA:  Inflammopharmacology     Country:  Switzerland    
Other Details:
Languages:  eng     Pagination:  233-40     Citation Subset:  IM    
Affiliation:
Research Center, New Jersey Dental School, University of Medicine and Dentistry of New Jersey, Newark, NJ 07103-2400, USA. slomiabr@umdnj.edu
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MeSH Terms
Descriptor/Qualifier:
Amidines / pharmacology
Animals
Apoptosis / drug effects*
Benzylamines / pharmacology
Cells, Cultured
Gastric Mucosa / cytology*,  drug effects,  metabolism
Ghrelin / metabolism*,  pharmacology
Helicobacter pylori / physiology*
Lipopolysaccharides / pharmacology
Nitric Oxide / metabolism
Nitric Oxide Synthase / metabolism*
Nitric Oxide Synthase Type II / antagonists & inhibitors,  metabolism
Phosphorylation
Rats
Chemical
Reg. No./Substance:
0/Amidines; 0/Benzylamines; 0/Ghrelin; 0/Lipopolysaccharides; 0/N-(3-(aminomethyl)benzyl)acetamidine; 10102-43-9/Nitric Oxide; EC 1.14.13.39/Nitric Oxide Synthase; EC 1.14.13.39/Nitric Oxide Synthase Type II

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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