Document Detail


Role of the commensal microbiota in normal and pathogenic host immune responses.
MedLine Citation:
PMID:  22018232     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The commensal microbiota that inhabit different parts of the gastrointestinal (GI) tract have been shaped by coevolution with the host species. The symbiotic relationship of the hundreds of microbial species with the host requires a tuned response that prevents host damage, e.g., inflammation, while tolerating the presence of the potentially beneficial microbes. Recent studies have begun to shed light on immunological processes that participate in maintenance of homeostasis with the microbiota and on how disturbance of host immunity or the microbial ecosystem can result in disease-provoking dysbiosis. Our growing appreciation of this delicate host-microbe relationship promises to influence our understanding of inflammatory diseases and infection by microbial pathogens and to provide new therapeutic opportunities.
Authors:
Dan R Littman; Eric G Pamer
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Review    
Journal Detail:
Title:  Cell host & microbe     Volume:  10     ISSN:  1934-6069     ISO Abbreviation:  Cell Host Microbe     Publication Date:  2011 Oct 
Date Detail:
Created Date:  2011-10-24     Completed Date:  2012-02-08     Revised Date:  2012-05-07    
Medline Journal Info:
Nlm Unique ID:  101302316     Medline TA:  Cell Host Microbe     Country:  United States    
Other Details:
Languages:  eng     Pagination:  311-23     Citation Subset:  IM    
Copyright Information:
Copyright © 2011 Elsevier Inc. All rights reserved.
Affiliation:
Molecular Pathogenesis Program, The Kimmel Center for Biology and Medicine of the Skirball Institute, New York University School of Medicine, New York, NY 10016, USA. dan.littman@med.nyu.edu
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MeSH Terms
Descriptor/Qualifier:
Animals
Gastrointestinal Tract / immunology*,  microbiology*
Humans
Metagenome / immunology*,  physiology*
Models, Biological
Symbiosis*
Grant Support
ID/Acronym/Agency:
R01 AI042135/AI/NIAID NIH HHS; R01 AI042135-15/AI/NIAID NIH HHS; R01 AI080619/AI/NIAID NIH HHS; RC2 AR058986/AR/NIAMS NIH HHS; RC2 AR058986-02/AR/NIAMS NIH HHS; //Howard Hughes Medical Institute

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