Document Detail


The role of the IL-12 cytokine family in directing T-cell responses in oral candidosis.
MedLine Citation:
PMID:  20981280     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Candida albicans is an opportunistic fungal pathogen that normally exists as a harmless commensal in humans. In instances where host debilitation occurs, Candida can cause a range of clinical infections, and whilst these are primarily superficial, effecting mucosal membranes, systemic infections can develop in severely immunocompromised individuals. The mechanism of host immunity during commensal carriage of C. albicans has been intensively studied. In this paper, we present the most recent information concerning host recognition of C. albicans leading to cytokine production and the subsequent T-cell responses generated in response to C. albicans. Particular focus is given to the role of the IL-12 cytokine family including IL-12, IL-23, IL-27, and IL-35, in host immunity to Candida. CD4(+) T-cells are considered crucial in the regulation of immunity and inflammation. In this regard, the role of Th1/2, helper cells, together with the recently identified Th17 and Treg cells in candidosis will be discussed. Understanding the detailed mechanisms that underlie host immunity to Candida not only will be of benefit in terms of the infections caused by this organism but could also be exploited in the development of therapeutic interventions for other diseases.
Authors:
Xiao-Qing Wei; Helen Rogers; Michael A O Lewis; David W Williams
Publication Detail:
Type:  Journal Article; Review     Date:  2010-10-24
Journal Detail:
Title:  Clinical & developmental immunology     Volume:  2011     ISSN:  1740-2530     ISO Abbreviation:  Clin. Dev. Immunol.     Publication Date:  2011  
Date Detail:
Created Date:  2010-10-28     Completed Date:  2011-05-24     Revised Date:  2011-07-25    
Medline Journal Info:
Nlm Unique ID:  101183692     Medline TA:  Clin Dev Immunol     Country:  England    
Other Details:
Languages:  eng     Pagination:  697340     Citation Subset:  IM    
Affiliation:
Cardiff University, Heath Park, UK. weix1@cardiff.ac.uk
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MeSH Terms
Descriptor/Qualifier:
Animals
Candidiasis, Oral / immunology*
Host-Pathogen Interactions
Humans
Immunity, Cellular*
Immunomodulation
Interleukin-12 / genetics,  immunology*
Mice
Multigene Family / immunology
Receptors, Pattern Recognition / immunology*
T-Lymphocytes / immunology*
Th1-Th2 Balance
Chemical
Reg. No./Substance:
0/Receptors, Pattern Recognition; 187348-17-0/Interleukin-12
Comments/Corrections

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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