Document Detail


A role for BAF57 in cell cycle-dependent transcriptional regulation by the SWI/SNF chromatin remodeling complex.
MedLine Citation:
PMID:  20460533     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The SWI/SNF complex is an ATP-dependent chromatin remodeling complex that plays pivotal roles in gene regulation and cell cycle control. In the present study, we explored the molecular functions of the BAF57 subunit of SWI/SNF in cell cycle control via transcriptional regulation of cell cycle-related genes. We affinity purified SWI/SNF from HeLa cells stably expressing FLAG-tagged BAF47/Ini1 with or without stable short hairpin RNA-mediated knockdown of BAF57. The subunit composition of the holo-SWI/SNF and BAF57-depleted SWI/SNF complexes from these cells was determined using a quantitative SILAC (stable isotope labeling by amino acids in cell culture)-based proteomic approach. Depletion of BAF57 resulted in a significant codepletion of BAF180 from the SWI/SNF complex without decreasing total cellular BAF180 levels. In biochemical assays of SWI/SNF activity, the holo-SWI/SNF and BAF57/BAF180-depleted SWI/SNF complexes exhibited similar activities. However, in cell proliferation assays using HeLa cells, knockdown of BAF57 resulted in an accumulation of cells in the G(2)-M phase, inhibition of colony formation, and impaired growth in soft agar. Knockdown of BAF57 also caused transcriptional misregulation of various cell cycle-related genes, especially genes involved in late G(2). Collectively, our results have identified a new role for BAF57 within the SWI/SNF complex that is required for (a) maintaining the proper subunit composition of the complex and (b) cell cycle progression through the transcriptional regulation of a subset of cell cycle-related genes.
Authors:
Nasun Hah; Annemieke Kolkman; Donald D Ruhl; W W M Pim Pijnappel; Albert J R Heck; H Th Marc Timmers; W Lee Kraus
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't     Date:  2010-05-11
Journal Detail:
Title:  Cancer research     Volume:  70     ISSN:  1538-7445     ISO Abbreviation:  Cancer Res.     Publication Date:  2010 Jun 
Date Detail:
Created Date:  2010-06-02     Completed Date:  2010-06-17     Revised Date:  2011-07-28    
Medline Journal Info:
Nlm Unique ID:  2984705R     Medline TA:  Cancer Res     Country:  United States    
Other Details:
Languages:  eng     Pagination:  4402-11     Citation Subset:  IM    
Copyright Information:
Copyright 2010 AACR.
Affiliation:
Department of Molecular Biology and Genetics and Graduate Field of Biochemistry, Molecular and Cell Biology, Cornell University, Ithaca, NY 14853, USA.
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MeSH Terms
Descriptor/Qualifier:
Cell Cycle / genetics*
Chromatin Immunoprecipitation
Chromosomal Proteins, Non-Histone / genetics*
DNA-Binding Proteins / genetics*
Gene Expression Regulation, Neoplastic
Hela Cells
Humans
Nuclear Proteins / genetics
Transcription Factors / genetics*
Transcription, Genetic
Grant Support
ID/Acronym/Agency:
DK058110/DK/NIDDK NIH HHS; R01 DK058110-06/DK/NIDDK NIH HHS; R01 DK058110-07/DK/NIDDK NIH HHS; R01 DK058110-08/DK/NIDDK NIH HHS; R01 DK058110-09/DK/NIDDK NIH HHS; R01 DK058110-10/DK/NIDDK NIH HHS
Chemical
Reg. No./Substance:
0/Chromosomal Proteins, Non-Histone; 0/DNA-Binding Proteins; 0/Nuclear Proteins; 0/PBRM1 protein, human; 0/SMARCE1 protein, human; 0/SWI-SNF-B chromatin-remodeling complex; 0/Transcription Factors
Comments/Corrections

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