Document Detail


Role of angiotensin II in the development of nephropathy and podocytopathy of diabetes.
MedLine Citation:
PMID:  21067505     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Diabetic kidney disease is the leading cause of end-stage renal disease worldwide. Podocytes are highly differentiated, pericyte-like cells that are essential for normal function of the kidney filter. Loss of podocytes is a hallmark of progressive kidney diseases including diabetic nephropathy. Podocytes are a direct target for angiotensin II - mediated injury by altered expression and distribution of podocyte proteins. Additionally, angiotensin II promotes podocyte injury indirectly by increasing calcium influx and production of reactive oxygen species. Notwithstanding the convincing rationale for angiotensin II blockade as a treatment modality, the incidence of diabetes-related end stage renal disease has increased steadily despite widespread use of angiotensin converting enzyme inhibitors (ACEIs) and angiotensin receptor blockers (ARBs). Recently published clinical trials have rekindled a debate on the safety and efficacy of dual blockade of the renin-angiotensin system (RAS). This review summarizes the rationale for blockade of angiotensin II as a therapeutic target in treating diabetic kidney disease, including the critical role played by podocytes. Recent relevant clinical trials on the role of RAS blockade in the treatment of diabetic kidney disease are discussed.
Authors:
Kirk N Campbell; Leopoldo Raij; Peter Mundel
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Publication Detail:
Type:  Journal Article; Review    
Journal Detail:
Title:  Current diabetes reviews     Volume:  7     ISSN:  1875-6417     ISO Abbreviation:  Curr Diabetes Rev     Publication Date:  2011 Jan 
Date Detail:
Created Date:  2010-12-29     Completed Date:  2011-04-27     Revised Date:  2011-07-08    
Medline Journal Info:
Nlm Unique ID:  101253260     Medline TA:  Curr Diabetes Rev     Country:  United Arab Emirates    
Other Details:
Languages:  eng     Pagination:  3-7     Citation Subset:  IM    
Affiliation:
Department of Medicine, Leonard Miller School of Medicine, University of Miami, 1580 NW 10th Avenue, Miami, FL 33136, USA. kcampbell@med.miami.edu
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MeSH Terms
Descriptor/Qualifier:
Angiotensin II / genetics,  metabolism,  physiology*
Animals
Diabetic Nephropathies / etiology*,  genetics,  pathology
Humans
Models, Biological
Oxidative Stress / physiology
Podocytes / pathology*
Chemical
Reg. No./Substance:
11128-99-7/Angiotensin II

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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