Document Detail


Retinoic acid from the meninges regulates cortical neuron generation.
MedLine Citation:
PMID:  19879845     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Extrinsic signals controlling generation of neocortical neurons during embryonic life have been difficult to identify. In this study we demonstrate that the dorsal forebrain meninges communicate with the adjacent radial glial endfeet and influence cortical development. We took advantage of Foxc1 mutant mice with defects in forebrain meningeal formation. Foxc1 dosage and loss of meninges correlated with a dramatic reduction in both neuron and intermediate progenitor production and elongation of the neuroepithelium. Several types of experiments demonstrate that retinoic acid (RA) is the key component of this secreted activity. In addition, Rdh10- and Raldh2-expressing cells in the dorsal meninges were either reduced or absent in the Foxc1 mutants, and Rdh10 mutants had a cortical phenotype similar to the Foxc1 null mutants. Lastly, in utero RA treatment rescued the cortical phenotype in Foxc1 mutants. These results establish RA as a potent, meningeal-derived cue required for successful corticogenesis.
Authors:
Julie A Siegenthaler; Amir M Ashique; Konstantinos Zarbalis; Katelin P Patterson; Jonathan H Hecht; Maureen A Kane; Alexandra E Folias; Youngshik Choe; Scott R May; Tsutomu Kume; Joseph L Napoli; Andrew S Peterson; Samuel J Pleasure
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Publication Detail:
Type:  In Vitro; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Cell     Volume:  139     ISSN:  1097-4172     ISO Abbreviation:  Cell     Publication Date:  2009 Oct 
Date Detail:
Created Date:  2009-11-02     Completed Date:  2009-12-03     Revised Date:  2013-02-27    
Medline Journal Info:
Nlm Unique ID:  0413066     Medline TA:  Cell     Country:  United States    
Other Details:
Languages:  eng     Pagination:  597-609     Citation Subset:  IM    
Affiliation:
Department of Neurology, Institute for Regenerative Medicine, University of California San Francisco, San Francisco, CA 94158, USA.
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MeSH Terms
Descriptor/Qualifier:
Animals
Forkhead Transcription Factors / genetics,  metabolism
Meninges / metabolism*
Mice
Neurogenesis*
Neurons / cytology*
Prosencephalon / cytology,  metabolism
Tretinoin / metabolism*
Grant Support
ID/Acronym/Agency:
AS1625//Autism Speaks; K02 MH074958-05/MH/NIMH NIH HHS; R01 DA017627-11A1/DA/NIDA NIH HHS; R01DA017627/DA/NIDA NIH HHS; R56 DA017627-11/DA/NIDA NIH HHS
Chemical
Reg. No./Substance:
0/Forkhead Transcription Factors; 0/Foxc1 protein, mouse; 302-79-4/Tretinoin
Comments/Corrections
Erratum In:
Cell. 2011 Aug 5;146(3):486

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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