Document Detail

Response of the vascular system to different modifications of haemofiltration and haemodialysis.
MedLine Citation:
PMID:  7243769     Owner:  NLM     Status:  MEDLINE    
In order to evaluate the reasons for the better tolerance of the cardiovascular system to body fluid removal in HF, different modifications of single-pass and recirculation HD as well as post-dilution HF were applied in 6 patients with stable chronic renal insufficiency under identical conditions of fluid removal, Curea and use of dialysers. A remarkable tolerance of the vascular system could be observed in HF as in HD when the Na+ concentration in the dialysis or diluting fluid was raised from 130 to 150mEq/L or when plasma osmotic pressure was stabilised by i.v. infusion of mannitol. The different buffers acetate and lactate did not influence the results specifically. Total peripheral resistance and plasma noradrenaline levels increased in HF but showed no changes in HD. Important factors causing the greater tolerance of the cardiovascular system in HF may be a more stable extracellular osmotic pressure, inducing a rapid refilling of the extracellular space, combined with an increasing total peripheral resistance.
E Quellhorst; B Schuenemann; U Hildebrand; Z Falda
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Publication Detail:
Type:  Journal Article; Research Support, U.S. Gov't, P.H.S.    
Journal Detail:
Title:  Proceedings of the European Dialysis and Transplant Association. European Dialysis and Transplant Association     Volume:  17     ISSN:  0071-2736     ISO Abbreviation:  Proc Eur Dial Transplant Assoc     Publication Date:  1980  
Date Detail:
Created Date:  1981-08-10     Completed Date:  1981-08-10     Revised Date:  2008-02-21    
Medline Journal Info:
Nlm Unique ID:  0355210     Medline TA:  Proc Eur Dial Transplant Assoc     Country:  ENGLAND    
Other Details:
Languages:  eng     Pagination:  197-204     Citation Subset:  IM    
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MeSH Terms
Kidney Diseases / physiopathology,  therapy
Renal Dialysis / methods*
Ultrafiltration / methods*
Vascular Resistance
Grant Support
N01 - AM - 8 - 2229/AM/NIADDK NIH HHS

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