Document Detail


Respiratory syncytial virus F and G proteins induce interleukin 1alpha, CC, and CXC chemokine responses by normal human bronchoepithelial cells.
MedLine Citation:
PMID:  20205592     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Human respiratory syncytial virus (RSV) is a ubiquitous respiratory virus that causes serious lower respiratory tract disease in infants and young children worldwide. Studies have shown that RSV infection modulates chemokine expression patterns, suggesting that particular cytokine expression profiles may be indicators of disease severity. In this study, we show that RSV F or G protein treatment of fully differentiated primary normal human bronchial epithelial cells induces apical and basolateral secretion of interleukin 8 (IL-8), interferon-inducible protein 10 (IP-10), monocyte chemotactic protein 1 (MCP-1), and RANTES (regulated on activation, normal T cell expressed and secreted). Purified RSV G (attachment) protein was shown to stimulate the secretion of interleukin 1alpha and RANTES, whereas purified F (fusion) protein elicited the production of IL-8, IP-10, and RANTES. Studies of ultraviolet-inactivated RSV showed that treatment of normal human bronchial epithelial cells induces apical IL-8, IP-10, and MCP-1 secretion independent of infection, suggesting that RSV proteins alone modify the chemokine response pattern, which may affect the early immune response before infection.
Authors:
Christine M Oshansky; James P Barber; Jackelyn Crabtree; Ralph A Tripp
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural    
Journal Detail:
Title:  The Journal of infectious diseases     Volume:  201     ISSN:  1537-6613     ISO Abbreviation:  J. Infect. Dis.     Publication Date:  2010 Apr 
Date Detail:
Created Date:  2010-03-15     Completed Date:  2010-04-08     Revised Date:  2012-01-11    
Medline Journal Info:
Nlm Unique ID:  0413675     Medline TA:  J Infect Dis     Country:  United States    
Other Details:
Languages:  eng     Pagination:  1201-7     Citation Subset:  AIM; IM    
Affiliation:
Department of Infectious Diseases, College of Veterinary Medicine, Center for Disease Intervention, University of Georgia, Athens, Georgia 30602, USA.
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MeSH Terms
Descriptor/Qualifier:
Adolescent
Bronchi / immunology,  virology*
Cells, Cultured
Chemokine CXCL10 / biosynthesis
Chemokines, CC / biosynthesis*
Chemokines, CXC / biosynthesis*
Epithelium / immunology,  virology
Gene Expression Regulation, Viral / physiology
Humans
Interleukin-1alpha / biosynthesis*
Interleukin-8 / biosynthesis
Male
Respiratory Syncytial Virus Infections / immunology
Respiratory Syncytial Viruses / immunology
Viral Fusion Proteins / pharmacology*
Grant Support
ID/Acronym/Agency:
R01 AI069275-03/AI/NIAID NIH HHS; R01 AI088744-03/AI/NIAID NIH HHS; R01-AI069275-03/AI/NIAID NIH HHS
Chemical
Reg. No./Substance:
0/Chemokine CXCL10; 0/Chemokines, CC; 0/Chemokines, CXC; 0/F protein, human respiratory syncytial virus; 0/G glycoprotein, Respiratory syncytial virus; 0/Interleukin-1alpha; 0/Interleukin-8; 0/Viral Fusion Proteins
Comments/Corrections

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