Document Detail


Renal Nerves, WNK4, Glucocorticoids, and Salt Transport.
MedLine Citation:
PMID:  21641543     Owner:  NLM     Status:  In-Data-Review    
Abstract/OtherAbstract:
Roles of the sympathetic nervous system versus kidney salt transporters in hypertension are debated. A study in Nature Medicine (Mu et al., 2011) shows that dietary salt excess, coupled with β-adrenergic stimulation, increases arterial pressure via glucocorticoid receptors and WNK4, suggesting interactions between these systems in the pathogenesis of hypertension.
Authors:
David H Ellison; Virginia L Brooks
Related Documents :
625613 - The effects of changes of maternal pao2 and paco2 on the fetal pao2 and paco2--in vivo ...
8527283 - The cardiovascular and subjective effects of thyrotropin releasing hormone (trh) and a ...
335933 - Influence of abdominal pressure and sympathetic vasoconstriction on the cardiovascular ...
Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  Cell metabolism     Volume:  13     ISSN:  1932-7420     ISO Abbreviation:  Cell Metab.     Publication Date:  2011 Jun 
Date Detail:
Created Date:  2011-06-06     Completed Date:  -     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  101233170     Medline TA:  Cell Metab     Country:  United States    
Other Details:
Languages:  eng     Pagination:  619-20     Citation Subset:  IM    
Copyright Information:
Copyright © 2011 Elsevier Inc. All rights reserved.
Affiliation:
Division of Nephrology and Hypertension, Oregon Health and Science University, Portland, OR 97239, USA; Department of Physiology and Pharmacology, Oregon Health and Science University, Portland, OR 97239, USA; Veterans Affairs Medical Center, Portland, OR 97239, USA.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  A Radical Role for TOR in Longevity.
Next Document:  Fine tuning our cellular factories: sirtuins in mitochondrial biology.