Document Detail


Remyelination occurs as extensively but more slowly in old rats compared to young rats following gliotoxin-induced CNS demyelination.
MedLine Citation:
PMID:  10498825     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Age is one of the many factors that influence remyelination following CNS demyelination, although it is not clear whether it is the extent or rate of remyelination that is affected. To resolve this issue we have compared remyelination in young and old adult rat CNS following gliotoxin-induced demyelination. Remyelination of areas of ethidium bromide-induced demyelination in the caudal cerebellar peduncle reached completion by 4 weeks in young adult rats (2 months) but was not complete until 9 weeks in old adult rats (9-12 months). We have also shown that remyelination of lysolecithin-induced demyelination in the spinal white matter of old adult rats (18 months) can be extensive, with longer survival times (8 weeks) than have previously been examined. Thus, it is the rate rather than the extent of remyelination that changes in the ageing CNS. These results have important implications for understanding the mechanisms of remyelination, indicating that remyelination need not occur rapidly for it to be extensive. The capacity for the process of remyelination to continue over many weeks must also be borne in mind when assessing remyelination-enhancement strategies either by transplantation or promotion of endogenous mechanisms.
Authors:
S A Shields; J M Gilson; W F Blakemore; R J Franklin
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Publication Detail:
Type:  Comparative Study; Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Glia     Volume:  28     ISSN:  0894-1491     ISO Abbreviation:  Glia     Publication Date:  1999 Oct 
Date Detail:
Created Date:  1999-11-22     Completed Date:  1999-11-22     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  8806785     Medline TA:  Glia     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  77-83     Citation Subset:  IM    
Copyright Information:
Copyright 1999 Wiley-Liss, Inc.
Affiliation:
Department of Clinical Veterinary Medicine, University of Cambridge, Cambridge, United Kingdom.
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MeSH Terms
Descriptor/Qualifier:
Aging / physiology*
Animals
Cerebellar Nuclei / pathology
Demyelinating Diseases / chemically induced,  pathology*
Ethidium
Female
Fluorescent Dyes
Gliotoxin / toxicity*
Immunosuppressive Agents / toxicity*
Kinetics
Lysophosphatidylcholines / toxicity
Myelin Sheath / physiology*
Rats
Rats, Sprague-Dawley
Spinal Cord / pathology
Chemical
Reg. No./Substance:
0/Fluorescent Dyes; 0/Immunosuppressive Agents; 0/Lysophosphatidylcholines; 3546-21-2/Ethidium; 67-99-2/Gliotoxin
Comments/Corrections
Erratum In:
Glia 2000 Jan 1;29(1):102

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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