Document Detail


Remodeling of cardiolipin by phospholipid transacylation.
MedLine Citation:
PMID:  14551214     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Mitochondrial cardiolipin (CL) contains unique fatty acid patterns, but it is not known how the characteristic molecular species of CL are formed. We found a novel reaction that transfers acyl groups from phosphatidylcholine or phosphatidylethanolamine to CL in mitochondria of rat liver and human lymphoblasts. Acyl transfer was stimulated by ADP, ATP, and ATP gamma S, but not by other nucleotides. Coenzyme A stimulated the reaction only in the absence of adenine nucleotides. Free fatty acids were not incorporated into CL under the same incubation condition. The transacylation required addition of exogenous CL or monolyso-CL, whereas dilyso-CL was not a substrate. Transacylase activity was decreased in lymphoblasts from patients with Barth syndrome (tafazzin deletion), and this was accompanied by drastic changes in the molecular composition of CL. In rat liver, where linoleic acid was the most abundant residue of CL, only linoleoyl groups were transferred into CL, but not oleoyl or arachidonoyl groups. We demonstrated complete remodeling of tetraoleoyl-CL to tetralinoleoyl-CL in rat liver mitochondria and identified the intermediates linoleoyl-trioleoyl-CL, dilinoleoyl-dioleoyl-CL, and trilinoleoyl-oleoyl-CL by high-performance liquid chromatography. The data suggest that CL is remodeled by acyl specific phospholipid transacylation and that tafazzin is an acyltransferase involved in this mechanism.
Authors:
Yang Xu; Richard I Kelley; Thomas J J Blanck; Michael Schlame
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2003-10-09
Journal Detail:
Title:  The Journal of biological chemistry     Volume:  278     ISSN:  0021-9258     ISO Abbreviation:  J. Biol. Chem.     Publication Date:  2003 Dec 
Date Detail:
Created Date:  2003-12-15     Completed Date:  2004-01-30     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  2985121R     Medline TA:  J Biol Chem     Country:  United States    
Other Details:
Languages:  eng     Pagination:  51380-5     Citation Subset:  IM    
Affiliation:
Department of Anesthesiology, New York University School of Medicine, New York, New York 10016, USA.
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MeSH Terms
Descriptor/Qualifier:
Acylation
Acyltransferases / metabolism*
Animals
Carbon Radioisotopes
Cardiolipins / metabolism*
Case-Control Studies
Humans
Lipid Metabolism, Inborn Errors / metabolism*
Lymphocytes / metabolism
Mitochondria, Liver / metabolism
Phosphatidylcholines / metabolism
Phosphatidylethanolamines / metabolism
Proteins / metabolism
Rats
Transcription Factors*
Chemical
Reg. No./Substance:
0/Carbon Radioisotopes; 0/Cardiolipins; 0/Phosphatidylcholines; 0/Phosphatidylethanolamines; 0/Proteins; 0/TAZ protein, human; 0/Transcription Factors; EC 2.3.-/Acyltransferases

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