Document Detail


Regulation of steady-state neutrophil homeostasis by macrophages.
MedLine Citation:
PMID:  20980680     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The timely clearance of apoptotic neutrophils from inflammation sites is an important function of macrophages; however, the role of macrophages in maintaining neutrophil homeostasis under steady-state conditions is less well understood. By conditionally deleting the antiapoptotic gene cellular FLICE-like inhibitory protein (C-FLIP) in myeloid cells, we have generated a novel mouse model deficient in marginal zone and bone marrow stromal macrophages. These mice develop severe neutrophilia, splenomegaly, extramedullary hematopoiesis, decreased body weight, and increased production of granulocyte colony-stimulating factor (G-CSF) and IL-1β, but not IL-17. c-FLIP(f/f) LysM-Cre mice exhibit delayed clearance of circulating neutrophils, suggesting that failure of macrophages to efficiently clear apoptotic neutrophils causes production of cytokines that drive excess granulopoiesis. Further, blocking G-CSF but not IL-1R signaling in vivo rescues this neutrophilia, suggesting that a G-CSF-dependent, IL-1β-independent pathway plays a role in promoting neutrophil production in mice with defective clearance of apoptotic cells.
Authors:
Claire Gordy; Heather Pua; Gregory D Sempowski; You-Wen He
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.     Date:  2010-10-27
Journal Detail:
Title:  Blood     Volume:  117     ISSN:  1528-0020     ISO Abbreviation:  Blood     Publication Date:  2011 Jan 
Date Detail:
Created Date:  2011-01-14     Completed Date:  2011-02-23     Revised Date:  2012-01-13    
Medline Journal Info:
Nlm Unique ID:  7603509     Medline TA:  Blood     Country:  United States    
Other Details:
Languages:  eng     Pagination:  618-29     Citation Subset:  AIM; IM    
Affiliation:
Department of Immunology, Duke University Medical Center, Durham, NC, USA.
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MeSH Terms
Descriptor/Qualifier:
Animals
Bone Marrow Cells / cytology,  immunology,  metabolism
CASP8 and FADD-Like Apoptosis Regulating Protein / deficiency,  genetics
Cell Survival
Gene Knock-In Techniques
Granulocyte Colony-Stimulating Factor / metabolism
Hematopoiesis / genetics,  immunology
Homeostasis / genetics,  immunology*
Inflammation / genetics,  immunology
Macrophages / immunology*,  metabolism
Mice
Mice, Knockout
Neutrophils / immunology*,  metabolism
Spleen / cytology,  immunology,  metabolism
Grant Support
ID/Acronym/Agency:
AI073947/AI/NIAID NIH HHS; R01 AI073947-04/AI/NIAID NIH HHS; R01 AI073947-05/AI/NIAID NIH HHS; UC6-AI058607/AI/NIAID NIH HHS
Chemical
Reg. No./Substance:
0/CASP8 and FADD-Like Apoptosis Regulating Protein; 0/Cflar protein, mouse; 143011-72-7/Granulocyte Colony-Stimulating Factor

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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