Document Detail


Regulation of myocardial triacylglycerol synthesis and metabolism.
MedLine Citation:
PMID:  14643794     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Studies showing a correlation of excess myocardial triacylglycerol stores with apoptosis, fibrosis, and contractile dysfunction indicate that dysregulation of triacylglycerol metabolism may contribute to cardiac disease. This review covers the regulation of heart triacylglycerol accumulation at the critical control points of fatty acid uptake, enzymes of triacylglycerol synthesis, lipolysis, and lipoprotein secretion. These pathways are discussed in the context of the central role myocardial triacylglycerol plays in cardiac energy metabolism and heart disease.
Authors:
Tal M Lewin; Rosalind A Coleman
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.; Review    
Journal Detail:
Title:  Biochimica et biophysica acta     Volume:  1634     ISSN:  0006-3002     ISO Abbreviation:  Biochim. Biophys. Acta     Publication Date:  2003 Nov 
Date Detail:
Created Date:  2003-12-03     Completed Date:  2004-01-21     Revised Date:  2007-11-14    
Medline Journal Info:
Nlm Unique ID:  0217513     Medline TA:  Biochim Biophys Acta     Country:  Netherlands    
Other Details:
Languages:  eng     Pagination:  63-75     Citation Subset:  IM    
Affiliation:
Department of Nutrition, School of Public Health, University of North Carolina, CB #7400, Pittsboro Street, Chapel Hill, NC 27599, USA.
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MeSH Terms
Descriptor/Qualifier:
Animals
Fatty Acids / metabolism
Humans
Lipolysis
Lipoproteins / biosynthesis,  secretion
Myocardium / metabolism*
Triglycerides / biosynthesis*,  metabolism*
Grant Support
ID/Acronym/Agency:
DK56598/DK/NIDDK NIH HHS; DK59931/DK/NIDDK NIH HHS; DK59935/DK/NIDDK NIH HHS
Chemical
Reg. No./Substance:
0/Fatty Acids; 0/Lipoproteins; 0/Triglycerides

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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