Document Detail


Regulation of murine telomere length via Rtel.
MedLine Citation:
PMID:  15588516     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Normal somatic cells have a finite replicative capacity. With each cell division, telomeres (the physical ends of linear chromosomes) progressively shorten until they reach a critical length, at which point the cells enter replicative senescence. Some cells can maintain telomere length by the action of the telomerase enzyme. A recent article described in detail the organization and function of Rtel, a murine gene encoding a DNA helicase-like protein. The Rtel protein was found to be essential for genomic stability and embryonic development and survival, and is proposed to be a dominant regulator of murine telomere length.
Authors:
Barry Flanary
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Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  Rejuvenation research     Volume:  7     ISSN:  1549-1684     ISO Abbreviation:  Rejuvenation Res     Publication Date:  2004  
Date Detail:
Created Date:  2004-12-13     Completed Date:  2005-02-01     Revised Date:  2007-09-11    
Medline Journal Info:
Nlm Unique ID:  101213381     Medline TA:  Rejuvenation Res     Country:  United States    
Other Details:
Languages:  eng     Pagination:  168-70     Citation Subset:  IM    
Affiliation:
Department of Neuroscience, McKnight Brain Institute, University of Florida College of Medicine, 100 Newell Dr., L1-100, Gainesville, FL 32610-0244, USA. beflana@ufl.edu
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MeSH Terms
Descriptor/Qualifier:
Animals
Cell Aging / genetics*
Cell Division
DNA Helicases / genetics*
Genes, Essential*
Genes, Regulator
Mice
Muridae / genetics
Telomere / physiology*
Chemical
Reg. No./Substance:
EC 3.6.1.-/DNA Helicases

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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