Document Detail


Regulation of mitochondrial gene expression by retinoids.
MedLine Citation:
PMID:  12440518     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Several nuclear hormone receptors have been localized to the mitochondrial compartment. Evidence supports the hypothesis that these receptors directly regulate mitochondrial transcription. Retinoic acid has also been shown to regulate mitochondrial transcription and function. This review discusses mechanisms of mitochondrial transcription and how retinoic acid may either indirectly or directly regulate mitochondrial transcription. How retinoic acid may affect individual nutrient requirements is also discussed.
Authors:
Helen B Everts; Carolyn D Berdanier
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.; Review    
Journal Detail:
Title:  IUBMB life     Volume:  54     ISSN:  1521-6543     ISO Abbreviation:  IUBMB Life     Publication Date:  2002 Aug 
Date Detail:
Created Date:  2002-11-20     Completed Date:  2003-07-01     Revised Date:  2008-11-21    
Medline Journal Info:
Nlm Unique ID:  100888706     Medline TA:  IUBMB Life     Country:  England    
Other Details:
Languages:  eng     Pagination:  45-9     Citation Subset:  IM    
Affiliation:
Department of Foods and Nutrition, University of Georgia, Athens 30602, USA.
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MeSH Terms
Descriptor/Qualifier:
Animals
Binding Sites
DNA, Mitochondrial / genetics,  metabolism*
Forecasting
Gene Expression Regulation / drug effects*
Humans
Models, Genetic
Promoter Regions, Genetic
Receptors, Retinoic Acid / drug effects,  genetics,  metabolism*
Receptors, Thyroid Hormone / metabolism
Transcription, Genetic
Tretinoin / metabolism,  pharmacology*
Chemical
Reg. No./Substance:
0/DNA, Mitochondrial; 0/Receptors, Retinoic Acid; 0/Receptors, Thyroid Hormone; 302-79-4/Tretinoin

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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