Document Detail


Regulation of fatty acid metabolism by cell autonomous circadian clocks: time to fatten up on information?
MedLine Citation:
PMID:  21296875     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Molecular, cellular, and animal-based studies have recently exposed circadian clocks as critical regulators of energy balance. Invariably, mouse models of genetically manipulated circadian clock components display features indicative of altered lipid/fatty acid metabolism, including differential adiposity and circulating lipids. The purpose of this minireview is to provide a comprehensive summary of current knowledge regarding the regulation of fatty acid metabolism by distinct cell autonomous circadian clocks. The implications of these recent findings for cardiometabolic disease and human health are discussed.
Authors:
Molly S Bray; Martin E Young
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Review     Date:  2011-02-04
Journal Detail:
Title:  The Journal of biological chemistry     Volume:  286     ISSN:  1083-351X     ISO Abbreviation:  J. Biol. Chem.     Publication Date:  2011 Apr 
Date Detail:
Created Date:  2011-04-04     Completed Date:  2011-07-19     Revised Date:  2012-04-09    
Medline Journal Info:
Nlm Unique ID:  2985121R     Medline TA:  J Biol Chem     Country:  United States    
Other Details:
Languages:  eng     Pagination:  11883-9     Citation Subset:  IM    
Affiliation:
Department of Epidemiology, University of Alabama at Birmingham, Birmingham, Alabama 35294, USA.
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MeSH Terms
Descriptor/Qualifier:
Animals
Circadian Clocks / genetics,  physiology*
Fatty Acids / metabolism*
Humans
Lipid Metabolism / genetics,  physiology*
Grant Support
ID/Acronym/Agency:
HL-074259/HL/NHLBI NIH HHS
Chemical
Reg. No./Substance:
0/Fatty Acids

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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