Document Detail


Reduced systolic pressure load decreases cell-cycle activity in the fetal sheep heart.
MedLine Citation:
PMID:  20484695     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The fetal heart is highly sensitive to changes in mechanical load. We have previously demonstrated that increased cardiac load can stimulate cell cycle activity and maturation of immature cardiomyocytes, but the effects of reduced load are not known. Sixteen fetal sheep were given either continuous intravenous infusion of lactated Ringer solution (LR) or enalaprilat, an angiotensin-converting enzyme inhibitor beginning at 127 days gestational age. After 8 days, fetal arterial pressure in the enalaprilat-infused fetuses (23.8 +/- 2.8 mmHg) was lower than that of control fetuses (47.5 +/- 4.7 mmHg) (P < 0.0001). Although the body weights of the two groups of fetuses were similar, the heart weight-to-body weight ratios of the enalaprilat-infused fetuses were less than those of the LR-infused fetuses (5.6 +/- 0.5 g/kg vs. 7.0 +/- 0.6 g/kg, P < 0.0001). Dimensions of ventricular myocytes were not different between control and enalaprilat-infused fetuses. However, there was a significant decrease in cell cycle activity in both the right ventricle (P < 0.005) and the left ventricle (P < 0.002) of the enalaprilat-infused fetuses. Thus, we conclude a sustained reduction in systolic pressure load decreases hyperplastic growth in the fetal heart.
Authors:
P F O'Tierney; D F Anderson; J J Faber; S Louey; K L Thornburg; G D Giraud
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.     Date:  2010-05-19
Journal Detail:
Title:  American journal of physiology. Regulatory, integrative and comparative physiology     Volume:  299     ISSN:  1522-1490     ISO Abbreviation:  Am. J. Physiol. Regul. Integr. Comp. Physiol.     Publication Date:  2010 Aug 
Date Detail:
Created Date:  2010-08-03     Completed Date:  2010-09-08     Revised Date:  2011-08-03    
Medline Journal Info:
Nlm Unique ID:  100901230     Medline TA:  Am J Physiol Regul Integr Comp Physiol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  R573-8     Citation Subset:  IM    
Affiliation:
Heart Research Center, Oregon Health and Sciences Univ., Portland, OR 97239, USA.
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MeSH Terms
Descriptor/Qualifier:
Angiotensin-Converting Enzyme Inhibitors / administration & dosage
Animals
Antihypertensive Agents / administration & dosage
Blood Pressure* / drug effects
Carbon Dioxide / blood
Cell Cycle* / drug effects
Cell Differentiation
Cell Size
Enalaprilat / administration & dosage
Fetal Heart / drug effects,  pathology*,  physiopathology
Fetal Weight
Gestational Age
Hyperplasia
Infusions, Intravenous
Myocytes, Cardiac / drug effects,  pathology*
Oxygen / blood
Sheep
Systole
Time Factors
Grant Support
ID/Acronym/Agency:
P01HD34430/HD/NICHD NIH HHS
Chemical
Reg. No./Substance:
0/Angiotensin-Converting Enzyme Inhibitors; 0/Antihypertensive Agents; 124-38-9/Carbon Dioxide; 7782-44-7/Oxygen; 84680-54-6/Enalaprilat
Comments/Corrections

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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