Document Detail


Recruitment of apoptotic cysteine proteases (caspases) in influenza virus-induced cell death.
MedLine Citation:
PMID:  10338194     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Influenza virus infection induces apoptosis in cultured cells with an augmented expression of Fas (APO-1/CD95). Caspases, a family of cysteine proteases structurally related to interleukin-1-beta-converting enzyme (ICE), play crucial roles in apoptosis induced by various stimuli, including Fas. However, activation of the caspase-cascade seems to be different in various pathways of apoptotic stimuli. We therefore examined the involvement of caspases in influenza virus-induced apoptosis using caspase inhibitors. We found that z-VAD-fmk and z-IETD-fmk effectively inhibited virus-induced apoptosis, whereas Ac-DEVD-CHO and Ac-YVAD-CHO showed partial and little effect on virus-induced cell death, respectively. Consistently, caspase-3-like activity, but not caspase-1-like activity, was increased in the virus-infected cells. The transfection of plasmids encoding viral inhibitors of caspase (v-FLIP or crmA) into HeLa cells inhibited apoptosis by virus infection. The peptide inhibitors of caspases used in this study did not inhibit viral replication. We conclude that influenza virus infection activates some caspases, and that this activation may be downstream of viral replication.
Authors:
T Takizawa; C Tatematsu; K Ohashi; Y Nakanishi
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Microbiology and immunology     Volume:  43     ISSN:  0385-5600     ISO Abbreviation:  Microbiol. Immunol.     Publication Date:  1999  
Date Detail:
Created Date:  1999-09-03     Completed Date:  1999-09-03     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  7703966     Medline TA:  Microbiol Immunol     Country:  JAPAN    
Other Details:
Languages:  eng     Pagination:  245-52     Citation Subset:  IM    
Affiliation:
Department of Biochemistry, Institute for Developmental Research, Aichi Human Service Center, Kasugai, Japan. takizawa@inst-hsc.pref.aichi.jp
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MeSH Terms
Descriptor/Qualifier:
Apoptosis*
Caspases / antagonists & inhibitors,  metabolism*
Enzyme Activation
Enzyme Inhibitors
Hela Cells
Humans
Influenza A virus / physiology*
Virus Replication
Chemical
Reg. No./Substance:
0/Enzyme Inhibitors; EC 3.4.22.-/Caspases

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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