Document Detail


Recovery of rat growth and lipid profiles in adult rats subjected to fetal protein malnutrition with a fructose-rich diet.
MedLine Citation:
PMID:  20227002     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
There is evidence suggesting an association between fructose consumption and the development of metabolic syndrome. In turn, protein malnutrition in utero is proposed to "program" the fetal tissues, making them more susceptible to nutritional associated disorders. To test this hypothesis, the present study was designed to analyze body growth and metabolic aspects of rats subjected to fetal protein malnutrition and subsequently fed a fructose-rich diet. Wistar rats were distributed into 4 groups: balanced (B) diet-B diet offered the entire experimental period; balanced diet/fructose-B diet until birth and fructose-rich diet (F-60% fructose) until adulthood; low-protein (L) diet/balanced-L diet until birth and B diet until adulthood; low-protein diet/fructose (F)-L diet until birth and F diet until adulthood. After nutritional recovery, there was a restoration of serum glucose, total protein, and albumin concentrations, which were reduced by fetal malnutrition, and a restoration of the liver glycogen and lipids contents, which were increased by fetal malnutrition. This restoration was independent of the diet adopted after birth. It was verified that the high fructose diet arrested body growth of the rats independently of the nutritional state during fetal life and was associated with weight reduction and decrease of the adipose in some regions of the body (P < .05). Moreover, the serum concentrations of triglycerides and total cholesterol, which are indicators of metabolic syndrome, rose in the rats that ingested the fructose-rich diet (P < .05). In summary, high consumption of fructose impairs body growth and alters the circulating lipids independently of the protein nutrition in utero.
Authors:
Lucieli T Cambri; Ana C Ghezzi; Carla Ribeiro; Rodrigo A Dalia; Maria A Rostom de Mello
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Nutrition research (New York, N.Y.)     Volume:  30     ISSN:  1879-0739     ISO Abbreviation:  Nutr Res     Publication Date:  2010 Feb 
Date Detail:
Created Date:  2010-03-15     Completed Date:  2010-08-04     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  8303331     Medline TA:  Nutr Res     Country:  United States    
Other Details:
Languages:  eng     Pagination:  156-62     Citation Subset:  IM    
Copyright Information:
Copyright 2010 Elsevier Inc. All rights reserved.
Affiliation:
Department of Physical Education, São Paulo State University (UNESP), Rio Claro-SP, Brazil. lucambri@yahoo.com.br
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Albumins / metabolism
Animals
Blood Glucose
Diet, Protein-Restricted
Dietary Sucrose / administration & dosage*
Female
Fetal Development / drug effects
Fetal Nutrition Disorders / diet therapy,  physiopathology*
Fructose / pharmacology*
Growth / drug effects*
Lipid Metabolism*
Liver Glycogen / metabolism
Male
Pregnancy
Prenatal Exposure Delayed Effects
Protein-Energy Malnutrition / diet therapy,  physiopathology*
Proteins / metabolism
Rats
Rats, Wistar
Weight Gain / drug effects*
Chemical
Reg. No./Substance:
0/Albumins; 0/Blood Glucose; 0/Dietary Sucrose; 0/Liver Glycogen; 0/Proteins; 30237-26-4/Fructose

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Stimulation of postprandial in vivo glycogenesis and lipogenesis of rats fed high fructose diet with...
Next Document:  Coronary blood flow and perfusion pressure during coronary angiography in patients with ongoing mech...