Document Detail


Receptor heteromerization and drug discovery.
MedLine Citation:
PMID:  20060175     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
G-protein-coupled receptors (GPCRs) are membrane proteins that convert extracellular information into intracellular signals. They are involved in many biological processes and therefore represent powerful targets to modulate physiological and pathological states. Recent studies have demonstrated that GPCR activity is regulated by several mechanisms. Among these, protein-protein interactions (and in particular interactions with other receptors leading to heteromerization) has been shown to have an important role in modulating GPCR function. This has expanded their repertoire of signaling and added a new level of regulation to their physiological roles. Emerging studies provide evidence for tissue-specific and disease-specific receptor heteromerization. This suggests that heteromers represent novel drug targets for the identification of selective compounds with potentially fewer side-effects.
Authors:
Raphael Rozenfeld; Lakshmi A Devi
Related Documents :
15258915 - Structural and functional characteristics of s1p receptors.
14567755 - Hormonal regulation of phospholipase cepsilon through distinct and overlapping pathways...
15817395 - How receptor mosaics decode transmitter signals. possible relevance of cooperativity.
22302025 - Tachyphylaxis to the sumatriptan-induced contractile effect in the human uterine artery...
10078615 - Resistance of gonadotropin-releasing hormone neurons to glutamatergic neurotoxicity.
21820405 - Islet-selectivity of g-protein coupled receptor ligands evaluated for pet imaging of pa...
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural     Date:  2010-01-07
Journal Detail:
Title:  Trends in pharmacological sciences     Volume:  31     ISSN:  1873-3735     ISO Abbreviation:  Trends Pharmacol. Sci.     Publication Date:  2010 Mar 
Date Detail:
Created Date:  2010-03-08     Completed Date:  2010-06-02     Revised Date:  2011-09-26    
Medline Journal Info:
Nlm Unique ID:  7906158     Medline TA:  Trends Pharmacol Sci     Country:  England    
Other Details:
Languages:  eng     Pagination:  124-30     Citation Subset:  IM    
Copyright Information:
2009 Elsevier Ltd. All rights reserved.
Affiliation:
Department of Pharmacology and Systems Therapeutics, Mount Sinai School of Medicine, One Gustave L. Levy Place, New York, NY10029, USA.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Drug Discovery / methods*
Ligands
Protein Binding
Protein Multimerization
Receptors, G-Protein-Coupled* / metabolism,  physiology
Signal Transduction
Grant Support
ID/Acronym/Agency:
1P50GM071558-01A27398/GM/NIGMS NIH HHS; AA017067/AA/NIAAA NIH HHS; DA08862/DA/NIDA NIH HHS; DA19521/DA/NIDA NIH HHS; K05 DA019521-05/DA/NIDA NIH HHS; R01 DA008863-15/DA/NIDA NIH HHS
Chemical
Reg. No./Substance:
0/Ligands; 0/Receptors, G-Protein-Coupled
Comments/Corrections

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  The role of valproate in metabolic disturbances in bipolar disorder patients.
Next Document:  Induction of oxidative stress and apoptosis by silver nanoparticles in the liver of adult zebrafish.