Document Detail


RHO methylation matters: a role for isoprenylcysteine carboxylmethyltransferase in cell migration and adhesion.
MedLine Citation:
PMID:  20798596     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Numerous proteins involved in diverse aspects of cell biology undergo a process of post-translational modification termed prenylation. The prenylation pathway consists of three enzymatic steps, the final of which is methylation of the carboxyl-terminal prenylcysteine formed in the first two steps by the enzyme isoprenylcysteine carboxylmethyltransferase (Icmt). Due to the prevalence of prenylated proteins in cancer biology, and the findings that several of the proteins are involved in processes controlling cell migration and adhesion, we sought to examine the role of Icmt - mediated methylation on cell behavior associated with metastasis. We found that inhibition of methylation reduces migration of the highly metastatic MDA-MB-231 breast cancer cell line. In addition, cell adhesion and cell spreading were also impaired by Icmt inhibition. Further investigation revealed that inhibition of Icmt significantly decreased the activation of both RhoA and Rac1, which are both prenylated proteins. The data obtained were consistent with the decreased activation being due to an increase in Rho GDP-dissociation inhibitor (GDI) binding by both proteins in the absence of their methylation. Importantly, the addition of exogenous RhoA or Rac1 to cells in which Icmt was inhibited was able to partially, but selectively, rescue directed and random migration, respectively. These results establish a role for Icmt-mediated methylation in cell migration, and point to specific prenylated proteins involved in this biology. The prenylation pathway has been targeted for oncogenic therapies, but the role of methylation in cell motility had been largely unexplored until now. The finding that methylation of Rho family members impacts on a specific component of their function provides an additional avenue through which to interrogate the biology of this important class of regulatory proteins.
Authors:
Ian Cushman; Patrick J Casey
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Publication Detail:
Type:  Journal Article     Date:  2011-01-01
Journal Detail:
Title:  Cell adhesion & migration     Volume:  5     ISSN:  1933-6926     ISO Abbreviation:  Cell Adh Migr     Publication Date:    2011 Jan-Feb
Date Detail:
Created Date:  2011-01-28     Completed Date:  2011-04-18     Revised Date:  2012-01-02    
Medline Journal Info:
Nlm Unique ID:  101469464     Medline TA:  Cell Adh Migr     Country:  United States    
Other Details:
Languages:  eng     Pagination:  11-5     Citation Subset:  IM    
Affiliation:
Department of Pharmacology and Cancer Biology, Duke University Medical Center, Durham, North Carolina, USA.
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MeSH Terms
Descriptor/Qualifier:
Cell Adhesion / physiology*
Cell Line, Tumor / chemistry
Cell Movement / physiology*
Guanine Nucleotide Dissociation Inhibitors / chemistry*
Humans
Methylation
Protein Binding
Protein Methyltransferases / chemistry*
Protein Prenylation / physiology
Protein Processing, Post-Translational / physiology
Signal Transduction / physiology
rac1 GTP-Binding Protein / chemistry
rho GTP-Binding Proteins / chemistry*
rhoA GTP-Binding Protein / chemistry
Chemical
Reg. No./Substance:
0/Guanine Nucleotide Dissociation Inhibitors; 133312-85-3/rho guanine nucleotide dissociation inhibitors; EC 2.1.1.-/Protein Methyltransferases; EC 2.1.1.100/protein-S-isoprenylcysteine O-methyltransferase; EC 3.6.5.2/rac1 GTP-Binding Protein; EC 3.6.5.2/rho GTP-Binding Proteins; EC 3.6.5.2/rhoA GTP-Binding Protein

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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