Document Detail


Quantitative theory of telomere length regulation and cellular senescence.
MedLine Citation:
PMID:  20207949     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
In normal somatic cells, telomere length shortens with each cell replication. This progressive shortening is associated with cellular senescence and apoptosis. Germ cells, stem cells, and the majority of cancer cells express telomerase, an enzyme that extends telomere length and, when expressed at sufficient levels, can immortalize or extend the life span of a cell line. It is believed that telomeres switch between two states: capped and uncapped. The telomere state determines its accessibility to telomerase and also the onset of senescence. One hypothesis is that the t loop, a large lariat-like structure, represents the capped state. In this paper we model a telomere state on the basis of the biophysics of t-loop formation, allowing us to develop a single mathematical model that accounts for two processes: telomere length regulation for telomerase positive cells and cellular senescence in somatic cells. The model predicts the steady-state length distribution for telomerase positive cells, describes the time evolution of telomere length, and computes the life span of a cell line on the basis of the levels of TRF2 and telomerase expression. The model reproduces a wide range of experimental behavior and fits data from immortal cell lines (HeLa S3 and 293T) and somatic cells (human diploid fibroblasts) well. We conclude that the t loop as the capped state is a quantitatively reasonable model of telomere length regulation and cellular senescence.
Authors:
Ignacio A Rodriguez-Brenes; Charles S Peskin
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.     Date:  2010-03-05
Journal Detail:
Title:  Proceedings of the National Academy of Sciences of the United States of America     Volume:  107     ISSN:  1091-6490     ISO Abbreviation:  Proc. Natl. Acad. Sci. U.S.A.     Publication Date:  2010 Mar 
Date Detail:
Created Date:  2010-03-24     Completed Date:  2010-04-16     Revised Date:  2010-09-30    
Medline Journal Info:
Nlm Unique ID:  7505876     Medline TA:  Proc Natl Acad Sci U S A     Country:  United States    
Other Details:
Languages:  eng     Pagination:  5387-92     Citation Subset:  IM    
Affiliation:
Courant Institute of Mathematical Sciences, New York University, New York, NY 10012, USA. ignacio@cims.nyu.edu
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MeSH Terms
Descriptor/Qualifier:
Algorithms
Biophysical Phenomena
Cell Aging / physiology*
Cell Line
DNA Damage
DNA Replication
Hela Cells
Humans
Models, Biological*
Stochastic Processes
Telomere / chemistry,  genetics,  physiology*
Telomeric Repeat Binding Protein 2 / physiology
Grant Support
ID/Acronym/Agency:
P50GM071558/GM/NIGMS NIH HHS
Chemical
Reg. No./Substance:
0/TERF2 protein, human; 0/Telomeric Repeat Binding Protein 2
Comments/Corrections

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