Document Detail


Psychometric properties of the Norwegian MacNew Heart Disease health-related quality of life inventory.
MedLine Citation:
PMID:  20133205     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
BACKGROUND: MacNew is a health-related quality of life (HQOL) inventory for patients with ischaemic heart disease and includes three domains; emotional, physical and social. The MacNew gives new opportunities to compare directly patients with the three major ischaemic heart diagnoses, angina, myocardial infarction and heart failure. However, this inventory has not earlier been evaluated in Norwegian.
OBJECTIVE: The purpose of this study was to validate the Norwegian version of the disease-specific HQOL inventory MacNew by assessing the measurement model, internal consistent reliability and test-retest and convergent validity.
METHOD: Patients with ischaemic heart disease (n=124; angina pectoris, n=53; myocardial infarction, n=35; and heart failure; n=36) participated in this cross-sectional study. Patients completed the self-administered MacNew and the generic HQOL-instrument SF-36. The MacNew and the SF-36 were repeated 2-3 weeks later (test-retest) by 17 patients.
RESULTS: The psychometric properties were satisfactory to good. The construct validity assessed by factor analysis explained 61.5% of the variance. The internal consistent reliability was high (exceeded 0.90 in all domains). The test-retest analysis showed equivalent results demonstrating good reproducible properties. The convergent validity within each domain was evaluated with correlation analysis, demonstrating correlations of 0.74, 0.69 and 0.71 on the emotional, physical and social domains, respectively.
CONCLUSION: The Norwegian version of the MacNew inventory demonstrated satisfactory to good psychometric properties and can be recommended for evaluating HQOL for patients with heart diseases in clinical studies.
Authors:
Aud Hiller; Anne-Sofie Helvik; Stein Kaasa; Stig A Slørdahl
Related Documents :
3996435 - Nitrate therapy for ischaemic heart disease.
10608025 - Optimizing the treatment of unstable angina.
12747595 - The -308 g/a tumor necrosis factor-alpha gene dimorphism: a risk factor for unstable an...
9453515 - Neovascularization in atherectomy specimens from patients with unstable angina: implica...
3345775 - Bidirectional transvenous/subcutaneous defibrillation of ventricular fibrillation in do...
2520655 - Altered concentrations of terminal complement complexes, anaphylatoxins, and leukotrien...
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2010-02-04
Journal Detail:
Title:  European journal of cardiovascular nursing : journal of the Working Group on Cardiovascular Nursing of the European Society of Cardiology     Volume:  9     ISSN:  1873-1953     ISO Abbreviation:  Eur J Cardiovasc Nurs     Publication Date:  2010 Sep 
Date Detail:
Created Date:  2010-08-09     Completed Date:  2010-12-16     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  101128793     Medline TA:  Eur J Cardiovasc Nurs     Country:  Netherlands    
Other Details:
Languages:  eng     Pagination:  146-52     Citation Subset:  IM; N    
Copyright Information:
(c) 2010 European Society of Cardiology. Published by Elsevier B.V. All rights reserved.
Affiliation:
Department of Cardiology, St. Olav's University Hospital, 7006 Trondheim, Norway. aud.hiller@stolav.no
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Adult
Aged
Aged, 80 and over
Angina Pectoris
Cross-Sectional Studies
Factor Analysis, Statistical
Female
Health Status Indicators*
Heart Failure
Humans
Middle Aged
Myocardial Infarction
Myocardial Ischemia*
Norway
Psychometrics
Quality of Life*
Reproducibility of Results

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Pulsed high-dose dexamethasone versus standard prednisolone treatment for chronic inflammatory demye...
Next Document:  Potentiation of TLR9 responses for human na??ve B-cell growth through RP105 signaling.