Document Detail


Psoriasis: from pathogenesis to novel therapeutic approaches.
MedLine Citation:
PMID:  20846119     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Psoriasis is one of the commonest chronic inflammatory disorders. Its cause is unknown, but a wealth of studies indicate that the disease results from a complex and dynamic interplay between genetic and environmental factors that trigger an excessive inflammatory response in the skin. Dendritic cells and effector T-cells are central in the development of the psoriastic lesion, and cytokines produced by these cells stimulate keratinocytes to proliferate and increase the migration of inflammatory cells into the skin, promoting epidermal hyperplasia and inflammation. Understanding the immunology of the psoriatic plaque has led to new therapeutic options and novel candidates for immunomodulation, and has changed the ways psoriatic patients are managed.
Authors:
Giovanni Monteleone; Francesco Pallone; Thomas T MacDonald; Sergio Chimenti; Antonio Costanzo
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't; Review    
Journal Detail:
Title:  Clinical science (London, England : 1979)     Volume:  120     ISSN:  1470-8736     ISO Abbreviation:  Clin. Sci.     Publication Date:  2011 Jan 
Date Detail:
Created Date:  2010-09-17     Completed Date:  2010-11-23     Revised Date:  2011-05-05    
Medline Journal Info:
Nlm Unique ID:  7905731     Medline TA:  Clin Sci (Lond)     Country:  England    
Other Details:
Languages:  eng     Pagination:  1-11     Citation Subset:  IM    
Affiliation:
Gastroenterology Unit, Internal Medicine Department, University Tor Vergata of Rome, 00133 Rome, Italy. gi.monteleone@med.uniroma2.it
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MeSH Terms
Descriptor/Qualifier:
Cytokines / immunology
Dermatologic Agents / therapeutic use
Genetic Predisposition to Disease
Humans
Immunity, Innate
Immunosuppressive Agents / therapeutic use
Psoriasis / drug therapy,  etiology*,  immunology
T-Lymphocyte Subsets / immunology
Chemical
Reg. No./Substance:
0/Cytokines; 0/Dermatologic Agents; 0/Immunosuppressive Agents

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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