Document Detail


Proteoglycan synthesis is increased in cells with impaired clathrin-dependent endocytosis.
MedLine Citation:
PMID:  11148135     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Overexpression of a GTPase deficient dynamin mutant in HeLa dynK44A cells causes a block in clathrin-dependent endocytosis. When endocytosis is inhibited, these cells incorporate higher levels of [(35)S]sulfate into both cellular and secreted macromolecules and larger amounts of proteoglycans such as syndecan and perlecan are immunoprecipitated from [(35)S]sulfate-labelled lysates. Gel filtration and ion-exchange chromatography revealed that the increased [(35)S]sulfate incorporation into proteoglycans was not due to significant differences in size or density of negative charge of glycosaminoglycan chains attached to proteoglycan core proteins. On the other hand, measurements of the syndecan-1 mRNA level and of [(3)H]leucine-labelled perlecan after immunoprecipitation supported the idea that the increased [(35)S]sulfate incorporation into proteoglycans was due to a selective increase in the synthesis of proteoglycan core proteins. Interestingly, the activity of protein kinase C was increased in cells expressing mutant dynamin and inhibition of protein kinase C with BIM reduced the differences in [(35)S]sulfate incorporation between cells with normal and impaired clathrin-dependent endocytosis. Thus, the activation of protein kinase C observed upon inhibition of clathrin-dependent endocytosis may be responsible for the increased synthesis of proteoglycans.
Authors:
A Llorente; K Prydz; M Sprangers; G Skretting; S O Kolset; K Sandvig
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Journal of cell science     Volume:  114     ISSN:  0021-9533     ISO Abbreviation:  J. Cell. Sci.     Publication Date:  2001 Jan 
Date Detail:
Created Date:  2001-01-26     Completed Date:  2001-05-17     Revised Date:  2009-11-19    
Medline Journal Info:
Nlm Unique ID:  0052457     Medline TA:  J Cell Sci     Country:  England    
Other Details:
Languages:  eng     Pagination:  335-43     Citation Subset:  IM    
Affiliation:
Department of Biochemistry, The Norwegian Radium Hospital, Montebello, Norway.
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MeSH Terms
Descriptor/Qualifier:
Animals
Cell Line
Chondroitin ABC Lyase
Chromatography, Gel
Chromatography, Ion Exchange
Clathrin / genetics,  metabolism*
Cricetinae
Cyclic AMP / metabolism
Cyclic AMP-Dependent Protein Kinases / metabolism
Dynamins
Endocytosis / physiology*
Fibroblast Growth Factor 1 / metabolism
GTP Phosphohydrolases / genetics,  metabolism
Hela Cells
Humans
Leucine / metabolism
Protein Kinase C / metabolism
Proteoglycans / biosynthesis*,  isolation & purification
Sulfates / metabolism
Sulfur Radioisotopes
Transcription, Genetic
Transfection
Transferrin / metabolism
Tritium
Chemical
Reg. No./Substance:
0/Clathrin; 0/Proteoglycans; 0/Sulfates; 0/Sulfur Radioisotopes; 10028-17-8/Tritium; 104781-85-3/Fibroblast Growth Factor 1; 11096-37-0/Transferrin; 60-92-4/Cyclic AMP; 61-90-5/Leucine; EC 2.7.11.11/Cyclic AMP-Dependent Protein Kinases; EC 2.7.11.13/Protein Kinase C; EC 3.6.1.-/GTP Phosphohydrolases; EC 3.6.5.5/Dynamins; EC 4.2.2.20/Chondroitin ABC Lyase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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