Document Detail


Protein expression and intracellular localization of prostate apoptosis response-4 (Par-4) are associated with apoptosis induction in nasopharyngeal carcinoma cell lines.
MedLine Citation:
PMID:  17881119     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Prostate apoptosis response-4 (Par-4) is a proapoptotic gene that selectively induces cell death in most cancer cells. In addition to the increased percentage of apoptotic cells, caspase-3 activity, and poly (ADP-ribose) polymerase (PARP) cleavage, we demonstrate that elevated expression of Par-4 and nuclear entry resulted in apoptosis of nasopharyngeal carcinoma (NPC) cell lines either in serum deprivation or by ectopic overexpression of Par-4. Moreover, disassociation from the Par-4/Akt complex was correlated with the induced proapoptotic ability of Par-4. Therefore, our data suggest that the cytoplasmic localization and expression level of endogenous Par-4 in NPC cells are not sufficient to augment apoptosis.
Authors:
Jeng-Woei Lee; Kuei-Fang Lee; Hsue-Yin Hsu; Lee-Ping Hsu; Wen-Ling Shih; Yi-Chih Chu; Wei-Ting Hsiao; Po-Fan Liu
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2007-09-18
Journal Detail:
Title:  Cancer letters     Volume:  257     ISSN:  0304-3835     ISO Abbreviation:  Cancer Lett.     Publication Date:  2007 Nov 
Date Detail:
Created Date:  2007-10-15     Completed Date:  2008-01-03     Revised Date:  2009-11-19    
Medline Journal Info:
Nlm Unique ID:  7600053     Medline TA:  Cancer Lett     Country:  Ireland    
Other Details:
Languages:  eng     Pagination:  252-62     Citation Subset:  IM    
Affiliation:
Department and Institute of Life Science, College of Life Science, Tzu-Chi University, 701, Sec. 3, Chung-Yang Road, Hualien 97071, Taiwan. jwlee@mail.tcu.edu.tw
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MeSH Terms
Descriptor/Qualifier:
Apoptosis / drug effects,  genetics,  physiology*
Apoptosis Regulatory Proteins / genetics,  metabolism*
Blotting, Western
Caspase 3 / metabolism
Cell Line, Tumor
Cell Nucleus / metabolism
Cell Survival / drug effects,  genetics,  physiology
Culture Media, Serum-Free / pharmacology
Fluorescent Antibody Technique
Gene Expression Regulation, Neoplastic / drug effects
Humans
Intracellular Fluid / drug effects,  metabolism*
Nasopharyngeal Neoplasms / genetics,  metabolism,  pathology
Poly(ADP-ribose) Polymerases / metabolism
Protein Binding
Proto-Oncogene Proteins c-akt / metabolism
Proto-Oncogene Proteins c-bcl-2 / genetics,  metabolism
Reverse Transcriptase Polymerase Chain Reaction
Time Factors
Transfection
Chemical
Reg. No./Substance:
0/Apoptosis Regulatory Proteins; 0/Culture Media, Serum-Free; 0/Proto-Oncogene Proteins c-bcl-2; 0/prostate apoptosis response-4 protein; EC 2.4.2.30/Poly(ADP-ribose) Polymerases; EC 2.7.11.1/Proto-Oncogene Proteins c-akt; EC 3.4.22.-/Caspase 3

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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