Document Detail


Proprioceptive neuropathy affects normalization of the H-reflex by exercise after spinal cord injury.
MedLine Citation:
PMID:  19913536     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The H-reflex habituates at relatively low frequency (10 Hz) stimulation in the intact spinal cord, but loss of descending inhibition resulting from spinal cord transection reduces this habituation. There is a return towards a normal pattern of low-frequency habituation in the reflex activity with cycling exercise of the affected hind limbs. This implies that repetitive passive stretching of the muscles in spinalized animals and the accompanying stimulation of large (Group I and II) proprioceptive fibers has modulatory effects on spinal cord reflexes after injury. To test this hypothesis, we induced pyridoxine neurotoxicity that preferentially affects large dorsal root ganglia neurons in intact and spinalized rats. Pyridoxine or saline injections were given twice daily (IP) for 6 weeks and half of the spinalized animals were subjected to cycling exercise during that period. After 6 weeks, the tibial nerve was stimulated electrically and recordings of M and H waves were made from interosseous muscles of the hind paw. Results show that pyridoxine treatment completely eliminated the H-reflex in spinal intact animals. In contrast, transection paired with pyridoxine treatment resulted in a reduction of the frequency-dependent habituation of the H-reflex that was not affected by exercise. These results indicate that normal Group I and II afferent input is critical to achieve exercise-based reversal of hyper-reflexia of the H-reflex after spinal cord injury.
Authors:
Karen Ollivier-Lanvin; Benjamin E Keeler; Rachel Siegfried; John D Houlé; Michel A Lemay
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural     Date:  2009-11-11
Journal Detail:
Title:  Experimental neurology     Volume:  221     ISSN:  1090-2430     ISO Abbreviation:  Exp. Neurol.     Publication Date:  2010 Jan 
Date Detail:
Created Date:  2010-01-27     Completed Date:  2010-02-16     Revised Date:  2011-07-19    
Medline Journal Info:
Nlm Unique ID:  0370712     Medline TA:  Exp Neurol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  198-205     Citation Subset:  IM    
Affiliation:
Department of Neurobiology and Anatomy, Drexel University College of Medicine, Philadelphia, PA 19129, USA.
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MeSH Terms
Descriptor/Qualifier:
Analysis of Variance
Animals
Disease Models, Animal
Electric Stimulation / methods
Electromyography / methods
Exercise Therapy / methods*
Female
Ganglia, Spinal / pathology
H-Reflex / physiology*
Nerve Fibers, Myelinated / drug effects,  pathology,  physiology*
Neurons / drug effects
Neurotoxicity Syndromes / etiology,  pathology,  physiopathology
Proprioception / physiology*
Pyridoxine / adverse effects,  pharmacology
Rats
Rats, Sprague-Dawley
Rats, Wistar
Reaction Time / drug effects,  physiology
Spinal Cord Injuries / pathology,  physiopathology*,  rehabilitation*
Grant Support
ID/Acronym/Agency:
NS007440/NS/NINDS NIH HHS; P01 NS055976/NS/NINDS NIH HHS; P01 NS055976-03/NS/NINDS NIH HHS; P01 NS055976-030003/NS/NINDS NIH HHS
Chemical
Reg. No./Substance:
65-23-6/Pyridoxine
Comments/Corrections

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