Document Detail


Promigratory activity of oxytocin on umbilical cord blood-derived mesenchymal stem cells.
MedLine Citation:
PMID:  20624160     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Recent studies show that oxytocin has various effects on cellular behaviors. Oxytocin is reported to stimulate cardiomyogenesis of embryonic stem cells and endothelial cell proliferation. Mesenchymal stem cells (MSCs) are widely used for cardiac repair, and we elucidated the effect of oxytocin on umbilical cord derived-MSCs (UCB-MSCs). UCB-MSCs were pretreated with oxytocin (100 nM) and washed with saline prior to experiments. To evaluate their angiogenic potential and migration activity, tube formation assay and Boyden chamber assay were performed. For in vivo study, ischemia-reperfusion was induced in rats, and UCB-MSCs with or without oxytocin pretreatment were injected into the infarcted myocardium to evaluate the engraftment of injected cells. Histological and hemodynamic studies were performed. Oxytocin-treated UCB-MSCs showed a decrease in tube formation but a drastic increase in transwell migration activity. The transcription level of matrix metalloproteinase (MMP)-2 was increased in oxytocin-treated UCB-MSCs. Knock-down of MMP-2 by use of siRNA restored the tube formation, while reducing transmigration activity. In rats injected with oxytocin-treated UCB-MSCs, cardiac fibrosis and CD68 infiltration in the peri-infarct zone were reduced, whereas cell engraftment and connexin43 expression were greater than in rats injected with untreated UCB-MSCs. By contrast, angiogenesis did not differ significantly between the two groups. Cardiac contractility was higher in the group injected with oxytocin-treated UCB-MSCs than in the group injected with phosphate-buffered saline alone. Collectively, oxytocin is an effective priming reagent for stem cells for application to damaged heart tissue.
Authors:
Yong Sook Kim; Jin Sook Kwon; Moon Hwa Hong; Jin Kim; Chang Hun Song; Myung Ho Jeong; Jeong Gwan Cho; Jong Chun Park; Jung Chae Kang; Youngkeun Ahn
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Artificial organs     Volume:  34     ISSN:  1525-1594     ISO Abbreviation:  Artif Organs     Publication Date:  2010 Jun 
Date Detail:
Created Date:  2010-07-13     Completed Date:  2010-10-18     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  7802778     Medline TA:  Artif Organs     Country:  United States    
Other Details:
Languages:  eng     Pagination:  453-61     Citation Subset:  IM    
Affiliation:
The Heart Center of Chonnam National University Hospital, 8 Hak Dong, Dong Ku, Gwangju 501-757, South Korea.
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MeSH Terms
Descriptor/Qualifier:
Animals
Cell Movement*
Cells, Cultured
Fetal Blood / cytology*
Gene Knockdown Techniques
Male
Matrix Metalloproteinase 2 / genetics,  metabolism
Mesenchymal Stem Cell Transplantation*
Mesenchymal Stem Cells / cytology*
Oxytocin / metabolism*
RNA, Small Interfering / genetics
Rats
Rats, Sprague-Dawley
Reperfusion Injury / therapy*
Chemical
Reg. No./Substance:
0/RNA, Small Interfering; 50-56-6/Oxytocin; EC 3.4.24.24/Matrix Metalloproteinase 2

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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