Document Detail


Proliferative effect of Apidra (insulin glulisine), a rapid-acting insulin analogue on mammary epithelial cells.
MedLine Citation:
PMID:  19480564     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The structural modification of insulin results in the generation of insulin analogues that show altered binding affinities to the insulin receptor and/or the IGF-I receptor, and as a consequence insulin analogues may have altered mitogenic potency. We analysed the proliferative effect of the rapid-acting insulin analogue Apidra (insulin glulisine) on mammary epithelial cells. We show that Apidra and Actrapid (recombinant human insulin) have similar proliferative effects on benign MCF10A and tumorigenic MCF7 cells and on epithelial cells of mouse mammary gland. Whereas Apidra and Actrapid induced similar activation of Erk1/2, activation of Akt/PKB by Apidra was significantly weaker compared to regular insulin. As AKT/PKB, an effector of the phosphoinositide 3-kinase pathway, mediates metabolic effects of insulin, we studied induction of hexokinase-2 in MCF7 cells and hexokinase-2 and hexokinase-4 in HepG2 cells by Actrapid and Apidra. Both genes were not significantly induced by Actrapid and Apidra in these cell lines.
Authors:
Ashish Shukla; Harald Enzmann; Doris Mayer
Related Documents :
3530724 - Demonstration of insulin receptors and modulation of alkaline phosphatase activity by i...
8550844 - Human diabetes associated with defects in nuclear regulatory proteins for the insulin r...
6380594 - Effects of membrane lipid unsaturation on the interactions of insulin and multiplicatio...
6303874 - Insulin binding to isolated monocytes and adipocytes in the study of hypoglycemic subje...
11168324 - Incidence and risk factors for neonatal hypoglycaemia among women with gestational diab...
10084574 - Hyperenterostatinemia in premenopausal obese women.
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Archives of physiology and biochemistry     Volume:  115     ISSN:  1744-4160     ISO Abbreviation:  Arch. Physiol. Biochem.     Publication Date:  2009 Jul 
Date Detail:
Created Date:  2009-07-29     Completed Date:  2010-01-05     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  9510153     Medline TA:  Arch Physiol Biochem     Country:  England    
Other Details:
Languages:  eng     Pagination:  119-26     Citation Subset:  IM    
Affiliation:
Hormones and Signal Transduction, German Cancer Research Centre, DKFZ-ZMBH Alliance, Heidelberg, Germany.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Animals
Cattle
Cell Line
Cell Proliferation / drug effects
Epithelial Cells / cytology*,  drug effects,  pathology*
Female
Humans
Insulin / analogs & derivatives*,  genetics,  metabolism,  pharmacology*
Mammary Glands, Animal / cytology*,  drug effects
Mammary Glands, Human / cytology*,  drug effects
Receptor, IGF Type 1 / metabolism
Recombinant Proteins / metabolism,  pharmacology
Chemical
Reg. No./Substance:
0/Recombinant Proteins; 11061-68-0/Insulin; EC 2.7.10.1/Receptor, IGF Type 1

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  PRAS40: target or modulator of mTORC1 signalling and insulin action?
Next Document:  Effects of omega-3 and omega-6 fatty acids on IGF-I receptor signalling in colorectal cancer cells.