Document Detail


Prokineticin-1: a novel mediator of the inflammatory response in third-trimester human placenta.
MedLine Citation:
PMID:  18372330     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Prokineticin-1 (PK1) is a recently described protein with a wide range of functions, including tissue-specific angiogenesis, modulation of inflammatory responses, and regulation of hemopoiesis. The aim of this study was to investigate the localization and expression of PK1 and PK receptor-1 (PKR1), their signaling pathways, and the effect of PK1 on expression of the inflammatory mediators cyclooxygenase (COX)-2 and IL-8 in third-trimester placenta. PK1 and PKR1 were highly expressed in term placenta and immunolocalized to syncytiotrophoblasts, cytotrophoblasts, fetal endothelium, and macrophages. PK1 induced a time-dependent increase in expression of IL-8 and COX-2, which was significantly reduced by inhibitors of Gq, cSrc, epidermal growth factor receptor (EGFR), and MAPK kinase. Treatment of third-trimester placenta with 40 nm PK1 induced a rapid phosphorylation of cSrc, EGFR, and ERK1/2. Phosphorylation of ERK1/2 in response to PK1 was dependent on sequential phosphorylation of cSrc and EGFR. Using double-immunofluorescent immunohistochemistry, PKR1 colocalized with IL-8 and COX-2 in placenta. These data suggest that PK1 may have a novel role as a mediator of the inflammatory response in placenta.
Authors:
Fiona C Denison; Sharon Battersby; Anne E King; Michael Szuber; Henry N Jabbour
Publication Detail:
Type:  In Vitro; Journal Article; Research Support, Non-U.S. Gov't     Date:  2008-03-27
Journal Detail:
Title:  Endocrinology     Volume:  149     ISSN:  0013-7227     ISO Abbreviation:  Endocrinology     Publication Date:  2008 Jul 
Date Detail:
Created Date:  2008-06-30     Completed Date:  2008-09-22     Revised Date:  2009-11-19    
Medline Journal Info:
Nlm Unique ID:  0375040     Medline TA:  Endocrinology     Country:  United States    
Other Details:
Languages:  eng     Pagination:  3470-7     Citation Subset:  AIM; IM    
Affiliation:
Department of Reproductive and Developmental Sciences, Centre for Reproductive Biology, University of Edinburgh, Edinburgh, United Kingdom.
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MeSH Terms
Descriptor/Qualifier:
Antigens, CD / metabolism
Antigens, CD31 / metabolism
Antigens, Differentiation, Myelomonocytic / metabolism
Blotting, Western
Cyclooxygenase 2 / metabolism
Extracellular Signal-Regulated MAP Kinases / metabolism
Female
Gene Expression / drug effects
Humans
Immunohistochemistry
Interleukin-8 / metabolism
Keratins / metabolism
Microscopy, Confocal
Microscopy, Fluorescence
Myometrium / metabolism
Phosphorylation / drug effects
Placenta / drug effects,  metabolism*
Pregnancy
Pregnancy Trimester, Third*
Receptor, Epidermal Growth Factor / metabolism
Receptors, G-Protein-Coupled / genetics,  metabolism
Reverse Transcriptase Polymerase Chain Reaction
Vascular Endothelial Growth Factor, Endocrine-Gland-Derived / genetics,  metabolism*,  pharmacology
Chemical
Reg. No./Substance:
0/Antigens, CD; 0/Antigens, CD31; 0/Antigens, Differentiation, Myelomonocytic; 0/CD68 antigen, human; 0/Interleukin-8; 0/PROKR1 protein, human; 0/Receptors, G-Protein-Coupled; 0/Vascular Endothelial Growth Factor, Endocrine-Gland-Derived; 68238-35-7/Keratins; EC 1.14.99.1/Cyclooxygenase 2; EC 1.14.99.1/PTGS2 protein, human; EC 2.7.10.1/Receptor, Epidermal Growth Factor; EC 2.7.11.24/Extracellular Signal-Regulated MAP Kinases
Comments/Corrections

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