Document Detail


Progressive depletion of complexin II in a transgenic mouse model of Huntington's disease.
MedLine Citation:
PMID:  11145989     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Huntington's disease (HD) is an autosomal dominant neurodegenerative disorder characterized by motor, emotional and cognitive dysfunction. There is no treatment or cure for this disease, and after the onset of symptoms, usually in the fourth decade of life, there is an inexorable decline to death. In many patients there is a complex deterioration of function before the onset of neuronal loss and, at least in mouse models, abnormalities in neurotransmission represent early events in the development of the disease. Here we describe the specific and progressive loss of complexin II from the brains of mice carrying the HD mutation (R6/2 line), and the later appearance of this protein in a subpopulation of neuronal intranuclear inclusions. Although the precise role of complexin II is still unclear, it is known to bind to the SNARE complex, and is therefore likely to be involved in the control of exocytosis. Our results suggest that changes in neurotransmitter release might contribute to the neuronal dysfunction seen in these mice.
Authors:
A J Morton; J M Edwardson
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Journal of neurochemistry     Volume:  76     ISSN:  0022-3042     ISO Abbreviation:  J. Neurochem.     Publication Date:  2001 Jan 
Date Detail:
Created Date:  2001-01-23     Completed Date:  2001-02-08     Revised Date:  2009-11-19    
Medline Journal Info:
Nlm Unique ID:  2985190R     Medline TA:  J Neurochem     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  166-72     Citation Subset:  IM    
Affiliation:
Department of Pharmacology, University of Cambridge, Cambridge, UK. ajm41@cam.ac.uk
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MeSH Terms
Descriptor/Qualifier:
Adaptor Proteins, Vesicular Transport
Aging / metabolism
Animals
Brain / metabolism,  pathology
Carrier Proteins / metabolism
Cell Nucleus / metabolism,  ultrastructure
Disease Models, Animal
Disease Progression
Huntington Disease / metabolism*,  pathology
Inclusion Bodies / metabolism
Membrane Proteins / metabolism
Mice
Mice, Transgenic
Nerve Tissue Proteins / deficiency*,  metabolism*
Organ Specificity
SNARE Proteins
Soluble N-Ethylmaleimide-Sensitive Factor Attachment Proteins
Synaptic Transmission
Synaptic Vesicles / metabolism
Vesicular Transport Proteins*
Chemical
Reg. No./Substance:
0/Adaptor Proteins, Vesicular Transport; 0/Carrier Proteins; 0/Membrane Proteins; 0/Nerve Tissue Proteins; 0/SNARE Proteins; 0/Soluble N-Ethylmaleimide-Sensitive Factor Attachment Proteins; 0/Vesicular Transport Proteins; 0/complexin II

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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