Document Detail


Pro-apoptotic effects of nivalenol and deoxynivalenol trichothecenes in J774A.1 murine macrophages.
MedLine Citation:
PMID:  19410639     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Nivalenol (NIV) and deoxynivalenol (DON) are trichothecenes mycotoxins produced by Fusarium fungi that occur in cereal grains alone or in combination. Several studies have shown that exposure to high concentrations of these mycotoxins resulted in decreased cell proliferation; however, the molecular mechanism underlying their activities are still partially known. In this study, we evaluated the effects of NIV and DON, alone and in combination, on J7741.A macrophages viability. The results of the current study show that both NIV and DON (10-100 microM) significantly stimulate apoptosis in J774A.1 macrophages in a concentration-dependent manner; in particular, NIV results a stronger pro-apoptotic effect than DON on cultured J774A.1 murine macrophages. No interactive effects were observed by exposing J774A.1 cells to both NIV and DON simultaneously. Pro-apoptotic activity induced by both mycotoxins seems to be essentially mediated by caspase-3 and is associated with a cell cycle blocking in G0/G1 phase. Moreover, our results show that NIV and DON are able to influence apoptotic pathway by ERK, pro-apoptotic protein Bax, caspase-3 and poly-ADP-ribose synthase (PARP), DNA repairing enzyme.
Authors:
Stefania Marzocco; Rosario Russo; Giuseppe Bianco; Giuseppina Autore; Lorella Severino
Related Documents :
10103099 - Cytochrome c release and caspase-3 activation during colchicine-induced apoptosis of ce...
10601269 - Survival and proliferation of cells expressing caspase-uncleavable poly(adp-ribose) pol...
19900759 - Tuning of apoptosis-mediator gene transcription in hepg2 human hepatoma cells through a...
20640889 - Caspase-3 triggers a tpck-sensitive protease pathway leading to degradation of the bh3-...
15788689 - Methylseleninic acid potentiates apoptosis induced by chemotherapeutic drugs in androge...
8626669 - D4-gdi, a substrate of cpp32, is proteolyzed during fas-induced apoptosis.
20534709 - An enigmatic tail of cd28 signaling.
10820249 - Gestational age-dependent expression of il-10 and its receptor in human placental tissu...
15889249 - The interplay between innate and adaptive immunity regulates cancer development.
Publication Detail:
Type:  Journal Article     Date:  2009-05-03
Journal Detail:
Title:  Toxicology letters     Volume:  189     ISSN:  1879-3169     ISO Abbreviation:  Toxicol. Lett.     Publication Date:  2009 Aug 
Date Detail:
Created Date:  2009-06-22     Completed Date:  2009-07-14     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  7709027     Medline TA:  Toxicol Lett     Country:  Netherlands    
Other Details:
Languages:  eng     Pagination:  21-6     Citation Subset:  IM    
Affiliation:
Department of Pharmaceutical Sciences, School of Pharmacy, University of Salerno, Fisciano (SA) 84084, Italy. smarzocco@unisa.it
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Animals
Apoptosis / drug effects*
Blotting, Western
Cell Cycle / drug effects
Cell Line
Cell Proliferation / drug effects
Cell Survival / drug effects
Dose-Response Relationship, Drug
Drug Interactions
Macrophages / drug effects*,  enzymology,  immunology,  metabolism
Mice
Mycotoxins / toxicity*
Trichothecenes / toxicity*
Chemical
Reg. No./Substance:
0/Mycotoxins; 0/Trichothecenes; 23282-20-4/nivalenol; 51481-10-8/deoxynivalenol

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Analyzing gene expression from relative codon usage bias in Yeast genome: a statistical significance...
Next Document:  Effects of in utero through lactational exposure to dicyclohexyl phthalate and p,p'-DDE in Sprague-D...