Document Detail


Prevalence and clinical significance of cardiovascular abnormalities in patients with the LEOPARD syndrome.
MedLine Citation:
PMID:  17697839     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The aim of this study was to characterize cardiovascular involvement in a large number of patients with LEOPARD syndrome. Twenty-six patients (age range 0 to 63 years, median age at the time of the study evaluation 17 years) underwent clinical and genetic investigations. Familial disease was ascertained in 9 patients. Nineteen patients (73%) showed electrocardiographic abnormalities. Left ventricular (LV) hypertrophy was present in 19 patients (73%), including 9 with LV outflow tract obstructions; right ventricular hypertrophy was present in 8 patients (30%). Valve (57%) and coronary artery (15%) anomalies were also observed. Single patients showed LV apical aneurysm, LV noncompaction, isolated LV dilation, and atrioventricular canal defect. During follow-up (9.1 +/- 4.5 years), 2 patients died suddenly, and 2 patients had cardiac arrest. These patients had LV hypertrophy. Despite the limited number of subjects studied, genotype-phenotype correlations were observed in familial cases. In conclusion, most patients with LEOPARD syndrome showed LV hypertrophy, often in association with other valvular or congenital defects. A spectrum of underrecognized cardiac anomalies were also observed. Long-term prognosis was benign, but the occurrence of 4 fatal events in patients with LV hypertrophy indicates that such patients require careful risk assessment and, in some cases, consideration for prophylaxis against sudden death.
Authors:
Giuseppe Limongelli; Giuseppe Pacileo; Bruno Marino; Maria Cristina Digilio; Anna Sarkozy; Perry Elliott; Paolo Versacci; Paolo Calabro; Andrea De Zorzi; Giovanni Di Salvo; Petros Syrris; Michael Patton; William J McKenna; Bruno Dallapiccola; Raffaele Calabro
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Publication Detail:
Type:  Journal Article; Multicenter Study; Research Support, Non-U.S. Gov't     Date:  2007-06-27
Journal Detail:
Title:  The American journal of cardiology     Volume:  100     ISSN:  0002-9149     ISO Abbreviation:  Am. J. Cardiol.     Publication Date:  2007 Aug 
Date Detail:
Created Date:  2007-08-16     Completed Date:  2007-09-27     Revised Date:  2007-11-15    
Medline Journal Info:
Nlm Unique ID:  0207277     Medline TA:  Am J Cardiol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  736-41     Citation Subset:  AIM; IM    
Affiliation:
Second University of Naples, Monaldi Hospital, Naples, Italy. limongelligiuseppe@libero.it
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MeSH Terms
Descriptor/Qualifier:
Adolescent
Adult
Cardiovascular Abnormalities* / complications,  epidemiology,  genetics
Child
Child, Preschool
DNA / genetics
Echocardiography
Electrocardiography, Ambulatory
Female
Follow-Up Studies
Genotype
Great Britain / epidemiology
Humans
Infant
Infant, Newborn
Intracellular Signaling Peptides and Proteins / genetics
Italy / epidemiology
LEOPARD Syndrome / complications*,  diagnosis,  genetics
Male
Middle Aged
Mutation
Polymerase Chain Reaction
Prevalence
Prognosis
Protein Tyrosine Phosphatase, Non-Receptor Type 11
Protein Tyrosine Phosphatases / genetics
Retrospective Studies
Risk Assessment
SH2 Domain-Containing Protein Tyrosine Phosphatases
Survival Rate
Time Factors
src Homology Domains
Chemical
Reg. No./Substance:
0/Intracellular Signaling Peptides and Proteins; 9007-49-2/DNA; EC 3.1.3.48/PTPN11 protein, human; EC 3.1.3.48/Protein Tyrosine Phosphatase, Non-Receptor Type 11; EC 3.1.3.48/Protein Tyrosine Phosphatases; EC 3.1.3.48/SH2 Domain-Containing Protein Tyrosine Phosphatases

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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