Document Detail


Preprotachykinin-A gene deletion protects mice against acute pancreatitis and associated lung injury.
MedLine Citation:
PMID:  12684214     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Impaired lung function in severe acute pancreatitis is the primary cause of morbidity and mortality in this condition. Preprotachykinin-A (PPT-A) gene products substance P and neurokinin (NK)-A have been shown to play important roles in neurogenic inflammation. Substance P acts primarily (but not exclusively) via the NK1 receptor. NKA acts primarily via the NK2 receptor. Earlier work has shown that knockout mice deficient in NK1 receptors are protected against acute pancreatitis and associated lung injury. NK1 receptors, however, bind other peptides in addition to substance P, not all of which are derived from the PPT-A gene. To examine the role of PPT-A gene products in acute pancreatitis, the effect of PPT-A gene deletion on the severity of acute pancreatitis and the associated lung injury was investigated. Deletion of PPT-A almost completely protected against acute pancreatitis-associated lung injury, with a partial protection against local pancreatic damage. These results show that PPT-A gene products are critical proinflammatory mediators in acute pancreatitis and the associated lung injury.
Authors:
Madhav Bhatia; John Slavin; Yuqing Cao; Allan I Basbaum; John P Neoptolemos
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  American journal of physiology. Gastrointestinal and liver physiology     Volume:  284     ISSN:  0193-1857     ISO Abbreviation:  Am. J. Physiol. Gastrointest. Liver Physiol.     Publication Date:  2003 May 
Date Detail:
Created Date:  2003-04-09     Completed Date:  2003-05-15     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  100901227     Medline TA:  Am J Physiol Gastrointest Liver Physiol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  G830-6     Citation Subset:  IM    
Affiliation:
Department of Pharmacology, National University of Singapore, Singapore 117597. mbhatia@nus.edu.sg
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MeSH Terms
Descriptor/Qualifier:
Acute Disease
Amylases / metabolism
Animals
Caerulein / pharmacology
Dose-Response Relationship, Drug
Gene Deletion*
Lung / metabolism,  pathology
Lung Diseases / complications*,  genetics*,  pathology
Mice
Mice, Knockout
Pancreas / metabolism,  pathology
Pancreatitis / complications*,  genetics*,  pathology
Protein Precursors / genetics*,  metabolism*
Tachykinins / genetics*,  metabolism*
Chemical
Reg. No./Substance:
0/Protein Precursors; 0/Tachykinins; 0/preprotachykinin; 17650-98-5/Caerulein; EC 3.2.1.-/Amylases

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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