Document Detail


Preischemic hyperglycemia leads to rapidly developing brain damage with no change in capillary patency.
MedLine Citation:
PMID:  9519261     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The present experiments were undertaken to explore whether exaggeration of ischemic brain damage by preischemic hyperglycemia is due to lack of capillary patency in the postischemic period. Normo- and hyperglycemic rats were exposed to 10 min of forebrain ischemia. Histopathological changes were evaluated after 6 and 16-18 h of recovery by light microscopy, and capillary patency was assessed at the same time points by a double-staining technique, depicting perfused and morphologically identifiable capillaries. The results demonstrate that some neuronal damage was detectable after 6 h of recirculation which was aggravated after 16-18 h of recirculation in hyperglycemic rats. In contrast, the degree of capillary patency was similar in normo- and hyperglycemic rats. In both groups the perfusion marker, Evans blue, perfused about 95% of all capillaries when injected 10 s before decapitation. Since preischemic hyperglycemia exaggerates brain damage without cessation of capillary perfusion the primary targets of hyperglycemic brain damage may not be capillaries but neurons or glial cells.
Authors:
P A Li; J Vogel; Q P He; M L Smith; W Kuschinsky; B K Siesjö
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.    
Journal Detail:
Title:  Brain research     Volume:  782     ISSN:  0006-8993     ISO Abbreviation:  Brain Res.     Publication Date:  1998 Jan 
Date Detail:
Created Date:  1998-05-14     Completed Date:  1998-05-14     Revised Date:  2007-11-14    
Medline Journal Info:
Nlm Unique ID:  0045503     Medline TA:  Brain Res     Country:  NETHERLANDS    
Other Details:
Languages:  eng     Pagination:  175-83     Citation Subset:  IM    
Affiliation:
Laboratory for Experimental Brain Research, Wallenberg Neuroscience Center, University of Lund, Sweden. lip@usask.sask.ca
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MeSH Terms
Descriptor/Qualifier:
Animals
Brain Damage, Chronic / etiology*,  pathology
Brain Ischemia / complications*,  pathology
Capillaries / pathology,  physiology
Cerebrovascular Circulation / physiology*
Hyperglycemia / complications*
Male
Rats
Rats, Wistar
Reperfusion
Time Factors
Vascular Patency / physiology*
Grant Support
ID/Acronym/Agency:
5 R01NS07838/NS/NINDS NIH HHS

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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