Document Detail


A Potential Role of Distinctively Delayed Blood Clearance of Recombinant Adeno-associated Virus Serotype 9 in Robust Cardiac Transduction.
MedLine Citation:
PMID:  21364543     Owner:  NLM     Status:  Publisher    
Abstract/OtherAbstract:
Recombinant adeno-associated virus serotype 9 (rAAV9) vectors show robust in vivo transduction by a systemic approach. It has been proposed that rAAV9 has enhanced ability to cross the vascular endothelial barriers. However, the scientific basis of systemic administration of rAAV9 and its transduction mechanisms have not been fully established. Here, we show indirect evidence suggesting that capillary walls still remain as a significant barrier to rAAV9 in cardiac transduction but not so in hepatic transduction in mice, and the distinctively delayed blood clearance of rAAV9 plays an important role in overcoming this barrier, contributing to robust cardiac transduction. We find that transvascular transport of rAAV9 in the heart is a capacity-limited slow process and occurs in the absence of caveolin-1, the major component of caveolae that mediate endothelial transcytosis. In addition, a reverse genetic study identifies the outer region of the icosahedral threefold capsid protrusions as a potential culprit for rAAV9's delayed blood clearance. These results support a model in which the delayed blood clearance of rAAV9 sustains the capacity-limited slow transvascular vector transport and plays a role in mediating robust cardiac transduction, and provide important implications in AAV capsid engineering to create new rAAV variants with more desirable properties.
Authors:
Nicole M Kotchey; Kei Adachi; Maliha Zahid; Katsuya Inagaki; Rakshita Charan; Robert S Parker; Hiroyuki Nakai
Related Documents :
9757923 - Effect of enteral versus parenteral feeding on hepatic blood flow and steady state prop...
15790103 - Continuous in vivo monitoring of blood diffusible calcium using on-line microdialysis s...
15563343 - Cognitive performance and cerebral blood flow in essential hypotension.
Publication Detail:
Type:  JOURNAL ARTICLE     Date:  2011-3-01
Journal Detail:
Title:  Molecular therapy : the journal of the American Society of Gene Therapy     Volume:  -     ISSN:  1525-0024     ISO Abbreviation:  -     Publication Date:  2011 Mar 
Date Detail:
Created Date:  2011-3-2     Completed Date:  -     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  100890581     Medline TA:  Mol Ther     Country:  -    
Other Details:
Languages:  ENG     Pagination:  -     Citation Subset:  -    
Affiliation:
Department of Microbiology and Molecular Genetics, University of Pittsburgh School of Medicine, Pittsburgh, Pennsylvania, USA.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Development of a Liver-specific Tet-On Inducible System for AAV Vectors and Its Application in the T...
Next Document:  Vascular gene transfer of SDF-1 promotes endothelial progenitor cell engraftment and enhances angiog...